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Go to irs.gov and use there programs to negotiate for a payment plan
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Beet This: Beetroot Juice Before Exercise Helps Older Brains Perform More Efficiently
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The level of your hunger could determine your bone structure
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Methionine-restricted diet, less meat, extends lifespan
Low amounts of the essential amino acid methionine diet help form new blood vessels
According to a new study from Harvard T.H. Chan School of Public Health, methionine-restricted diet extends lifespan and health span. Past work by Mitchell and associates has demonstrated that a methionine-limited diet routine builds production of the gas, hydrogen sulfide. Hydrogen sulfide, the gas that gives flatulence its repulsive smell, can help reduce the risk of heart attack, stroke, cancer and help stave off dementia, research suggests. When we are sick our cells create their own hydrogen sulfide which helps keep the mitochondria – our cell’s power generators – in working order. Methionine-restricted diet do NOT contains:
- Brazil Nuts
- Lean Beef & Lamb (Roast Beef)
- Cheese (Parmesan)
- Turkey & Chicken (Chicken Breast, cooked)
- Pork (Sirloin, cooked)
This smelly molecule, hydrogen sulfide, gives rotten eggs their characteristic odor, but is also made in our cells where it functions in myriad beneficial ways. One of these is to promote the growth of new blood vessels from endothelial cells—a process known as angiogenesis.
Lack of oxygen, or hypoxia, is the best-described trigger of angiogenesis. Hypoxia happens in tissues when a vessel is blocked, or upon intense exercise when oxygen delivery is constrained. Nonetheless, methionine restriction activated angiogenesis regardless of normal oxygen delivery, recommending involvement of a pathway detecting amino acid deprivation as opposed to hypoxia.
Large amounts of methionine can be found in eggs, meat, and fish; sesame seeds, Brazil nuts, and some other plant seeds; and oat grains.
Gut microbes and aging
The critical impact microbiota have on health and disease make the interaction between host and microbiome increasingly important as we evaluate therapeutics. Here we highlight growing evidence that beyond disease, microbes also affect the most fundamental of host physiological phenotypes, the rate of aging itself.
As you look in the mirror you may only see yourself staring back, but in reality you are not alone; you share your body with trillions of others. Contained on and within our bodies thrives a dynamic population of microbes that form a ‘metaorganism’, comprising 10 bacterial cells for every one of our own. Despite co-evolving in the presence of this ‘microbiome’ for 500 million years (Cho and Blaser, 2012), only recently have advances in sequencing technology allowed us to appreciate the complexities of this relationship, and the manner by which genomes within metaorganisms interact and affect one another. Inter-individual variations in the microbiome impact multiple human pathologies, from metabolic syndrome to cancer (Cho and Blaser, 2012). However, new data in invertebrate systems indicate microbes extend their effects beyond host pathology, to systemic modulation of the rate of aging.
The gut microbiome comprises a complex community of bacterial species that live within intestines of coelomate animals. Historically, the influence of gut microflora on host health has focused on the two extremes of the relationship: pathogenesis and symbiosis. Canonical examples of symbiotic host/microbe relationships include the reliance of some herbivores on gut flora for efficient digestion of cellulose. However, the majority of intestinal microbes are neither pathogenic nor classically symbiotic, but instead ‘commensal’, meaning their effect is non-harmful and neutral. Advances in high-throughput sequencing have facilitated the characterization of these diverse populations of commensal bacteria, defining the metagenome – the ensemble of host and microbiota DNA, and by extension the meta-transcriptome, proteome and metabolome. With endeavors such as the Human Microbiome Project just beginning (Cho and Blaser, 2012), research into the complexity of mammalian microbiome dynamics is in its infancy. Assigning causality to effects of the microbiome in mammalian systems is expensive, technically challenging and requires microbe-free husbandry conditions, and gnotobiotic mice with defined and controlled microbiota (Cho and Blaser, 2012).
Invertebrate model systems such as the fruit fly Drosophila melanogaster and the nematode worm Caenorhabditis elegans also live in the presence of microbiota both in nature and the laboratory (Ren et al., 2007; Zhang and Hou, 2013). Due to their genetic tractability and inexpensive husbandry, invertebrate studies allow direct causality to be assigned to the presence or absence of microbiota. Because of their short lifespans, D. melanogaster and C. elegans are also powerful models for studying genetic pathways that modulate the aging process. The ultimate goal of this research is to develop novel therapeutics with beneficial impacts on overall health in old age, complementing disease-centric approaches that focus on proximal determinants of individual pathologies. This work has uncovered conserved genes that modulate healthy aging in invertebrates and mammals, and are linked to extreme longevity in humans (Kenyon, 2010), including the insulin/IGF-1 like signaling (IIS) pathway, the target of rapamycin (TOR) and AMP-activated protein kinase (AMPK). However, little attention has been paid to how the microbial environment and the microbiome itself might affect the ability of these interventions to promote longevity. Here we review examples of microbiota in invertebrates directly and indirectly influencing the host genome to affect longevity, and discuss the impact these emerging data have on the field of aging research.
Direct Interspecies Signaling and Aging
Given the proximity of microbes and host within the metaorganism, diffusible molecules originating in bacteria can directly affect cells in the host (Ryan and Dow, 2008). Recent work suggests this interspecies signaling can impact host aging rate. C. elegans are commonly grown ‘monoxenically’ in co-culture with non-pathogenic strains of E. coli ‘OP50’ bacteria. E. coli act as nutrition for the worm, providing mandatory nutrients and essential components for life that the nematode cannot synthesize de novo. Although not considered pathogenic, E. coli OP50 do proliferate inside older animals, and raising C. elegans on non-dividing bacterial lawns suppresses this infection and increases worm lifespan (Zhang and Hou, 2013). In young worms however, E. coli are mechanically broken down before entering the gut and do not live in the animal as a true microbiome. Despite this, it is becoming apparent that beyond their role as a nutrient source and potential pathogen, E. coli secrete diffusible molecules, including metabolites and small RNAs that can directly impact C. elegans aging.
An example of a direct interspecies signal secreted by microbes that can promote longevity is nitric oxide (NO) (Gusarov et al., 2013). NO is a small, short-lived free radical that affects the activity of proteins both directly and indirectly via post-translational modifications. As a critical signaling molecule, NO has been implicated in multiple functions including neurotransmission, immunity and cardiovascular function (Gusarov et al., 2013). C. elegans are rare amongst eukaryotes as the cannot produce their own NO since they lack NO synthase. Gusarov et al. therefore speculated that worms might be utilizing NO produced from the bacteria within their microenvironment. Supporting their hypothesis, culturing C. elegans with NO-deficient Bacillus subtilis shortens their lifespan, while exogenous supplementation of NO increases it. Strikingly, an in vivo fluorescent sensor detected NO in multiple C. elegans tissues, suggesting it acts as a bona fide interspecies longevity signal. RNA-Seq analysis of worms fed NO identified specific transcriptional responses regulated by two transcription factors, DAF-16/FOXO and heat shock factor -1 (HSF-1), both known to mediate lifespan (Kenyon, 2010). Worms lacking DAF-16/FOXO or HSF-1 do not respond to the life prolonging effects of bacterial NO, meaning bacterial NO modulates C. elegans lifespan via effects on host transcription (Fig. 1A).
Microbe-modulation of invertebrate aging and physiology
A. Bacterial-derived signals NO and ncRNAs regulate C. elegans longevity. B. Metformin increases C. elegans lifespan via effects on bacterial folate metabolism. C. L. plantarum drive Drosophila growth under low nutrient conditions via the longevity modulator TOR. D. Different bacterial species elicit specific transcriptional responses in C. elegans.
California lawmakers pass sweeping rent cap bill
California lawmakers pass sweeping rent cap bill
Gov. Gavin Newsom is expected to sign the bill into law shortly
In a significant victory for California renters struggling to find affordable housing, state lawmakers on Wednesday sent a sweeping rent cap bill to the governor’s desk.
Assembly Bill 1482, which passed the Assembly Wednesday afternoon 46-22, is set to limit rent increases across the state to 5 percent plus inflation. Gov. Gavin Newsom, who helped broker a compromise between tenant advocacy groups and apartment owners, is expected to sign the bill into law in the coming days. The state joins Oregon, which passed similar legislation in February, and New York in enacting widespread rent caps.
“The question we have in front of us is what kind of a society do we want to live in?” said Assemblyman David Chiu (D-San Francisco), the bill’s lead author, just before the vote. “What kind of neighbors are we?”
In places like Oakland and San Francisco, which already have rent restriction, tenants may not notice much difference. And according to a recent Zillow analysis, the legislation would have benefited only around 7 percent of California renters if it had been in place last year, because rents in many places have ticked up just a few percentage points.
Still, Michael Lane, deputy director of the housing advocacy organization SV@Home, said the measure stands to have a sizable impact on cities that passed rent control measures a long time ago, like San Jose, because it will extend protections to newer apartments than have typically been covered. And in wealthy suburbs across the Bay Area, the new rent cap could have significant impact.
“It’s a historic day,” Lane said. “Hopes are high and this is one more step, but there’s still lots of work to do.”
Newsom praised lawmakers.
“In this year’s State of the State address, I asked the legislature to send me a strong renter protection package,” Newsom said in a statement. “Today, they sent me the strongest package in America. These anti-gouging and eviction protections will help families afford to keep a roof over their heads, and they will provide California with important new tools to combat our state’s broader housing and affordability crisis.”
AB 1482, which sunsets in 2030, will apply to apartments built at least 15 years ago, as well as houses and condos of the same age that are owned by large corporations. Tenant advocates also succeeded in pushing lawmakers to include just cause eviction protections for renters who have been in their home at least a year. That means landlords can’t kick out renters following the rules of their lease unless the owner is moving in, tearing down the residence, renovating it or taking it off the rental market.
Assemblywoman Buffy Wicks (D-Oakland) pushed back at the notion the measure will hamstring landlords.
“They still have a tremendous amount of flexibility to raise the rent,” Wicks said.
The measure does carve out exemptions for owners who live with their tenants, as well as owners who live in one part of a duplex and rent out the other unit. And landlords will be allowed to raise rents to market rates when a tenant moves out.
The measure is a significant political win for Newsom, who had taken a risk by backing the legislation before it had a clear path to passage. Last year, state voters shot down a ballot measure, Proposition 10, that would have strengthened local rent control policies across the state by eliminating the Costa-Hawkins Rental Housing Act, which restricts how far cities can go with rent control measures.While the California Apartment Association said it would remain neutral on the bill, the California Association of Realtors and most of the state’s Republican lawmakers opposed the measure, saying it would decrease the value of rental properties and deter developers from building more badly needed housing.
“We can build our way out of this if you allow it,” said Assemblywoman Melissa Melendez (R-Lake Elsinore).
Jared Martin, president of the California Association of Realtors, said in a statement after the vote he was disappointed.
“Although we did not prevail, we remain steadfast in our commitment to overcome California’s historic housing supply and affordability crisis,” Martin said. “Much more work remains ahead of us, and as we have said from the beginning, REALTORS® appreciate the commitment of Gov. Gavin Newsom, Democrats and Republicans to continue working to incentivize the production of new housing for rental and sale.”
But Democrats and renter advocacy groups have said the measure is necessary to stem a ballooning homelessness crisis and help families anticipate rent increases.
“Building more housing is going to take a lot of time,” said Assemblyman Al Muratsuchi (D-Torrance). “And in the meantime, in my community, even in affluent parts of my district, I’m hearing more and more stories of people that can’t keep up with their rents.”
What healthy breakfast foods have negative health effects in the long term?
What healthy breakfast foods have negative health effects in the long term?
Coffee. For seniors with ulcers, diabetes, gastritis or osteoporosis, the effects of caffeine may be more harmful and not worth the risk. If you do drink a cup in the morning, make it a fresh batch and add ginger. Ginger is good for pain, digestion, cleans the blood, a liver detox and excellent herb for healthy circulation.
How can drinking milk cause health issues?
How can drinking milk cause health issues?
Undigested fat from milk taxes the liver. Milk and other dairy products are the top source of saturated fat in the American diet, contributing to heart disease, type 2 diabetes, and Alzheimer’s disease. Studies have also linked dairy to an increased risk of breast, ovarian, and prostate cancers.
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Why high-dose vitamin C kills cancer cells
Why high-dose vitamin C kills cancer cells
Low levels of catalase enzyme make cancer cells vulnerable to high-dose vitamin C
- Date:
- January 9, 2017
- Source:
- University of Iowa Health Care
- Summary:
- Cancer researchers have homed in on how high-dose vitamin C kills cancer cells. Vitamin C breaks down to generate hydrogen peroxide, which can damage tissue and DNA. The new study shows that tumor cells with low levels of catalase enzyme activity are much less capable of removing hydrogen peroxide than normal cells, and are more susceptible to damage and death when they are exposed to high doses of vitamin C.
Vitamin C is an antioxidant and helps prevent oxidative stress.
In another study, vitamin C can target three vulnerabilities many cancer cells share: redox imbalance, epigenetic reprogramming and oxygen-sensing regulation. Although the mechanisms and predictive biomarkers that we discuss need to be validated in well-controlled clinical trials, these new discoveries regarding the anticancer properties of vitamin C are promising to help identify patient populations that may benefit the most from high-dose vitamin C therapy, developing effective combination strategies and improving the overall design of future vitamin C clinical trials for various types of cancer.
The same old tropes are there, from the claim that vitamin C has usefulness in treating cancer to the old ascorbate warriors’ lament that there’s no patent potential in vitamin C, which means that pharmaceutical companies don’t want to invest money into doing science and clinical trials on it because there’s no profit potential. Of course, I’ve written fairly extensively about vitamin C and cancer before, using it as an example of how even a two-time Nobel Prize winner like Linus Pauling could fall prey to bad science when he wandered outside of his area of expertise. Every so often these stories come up suggesting that Linus Pauling has somehow been vindicated and how vitamin C is the greatest thing for cancer patients since surgeons first discovered that some cancers could be cured by cutting them out. Inevitably, I have to throw cold water on such claims. No, Linus Pauling has not been vindicated, and, no, vitamin C for cancer is not all that great.
SJ Mayor Proposes ‘Granny Unit’ Program to Boost Housing Supply
SJ Mayor Proposes ‘Granny Unit’ Program to Boost Housing Supply
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Sam Liccardo wants to offer financial incentives for San Jose homeowners who build accessory dwelling units on their property.
San Jose Mayor Sam Liccardo pitched a creative idea to up the affordable housing stock, and some residents might already have the solution on their property: their garages.
Liccardo, in partnership with Housing Trust Silicon Valley, announced a program to waive city impact fees and offer loan incentives for San Jose homeowners who build an accessory dwelling unit (ADU), otherwise known as a “granny unit,” on their property.
A granny unit is an additional room built into an existing home. It can be a standalone unit or a garage, kitchen or similar space that has been converted into a housing unit.
The program, coined “Yes in My Backyard,” or YIMBY—the opposite of NIMBY, or Not in My Backyard—proposes an allocation of up to $5 million in housing funds for the loans, along with some matching grants from Housing Trust.
To participate, interested homeowners would agree to restrict their ADU for low- to mid-income households. In return, the city would issue a forgivable loan to cover construction and other related costs up to $20,000, so long as the homeowners keep their ADU affordable for at least five years and not put list their unit on Airbnb or any other similar short-term rent service during the duration of the loan.
“Our housing crisis requires we pursue creative and innovative approaches to spur more housing development at all income levels,” Liccardo said in a press release. “This unique opportunity to partner with the Housing Trust will help us leverage the city’s affordable housing dollars more efficiently, and increase the stock of affordable housing for families struggling to keep up with the high cost of living in our valley.”
Liccardo’s memo also suggested the YIMBY project could work with philanthropic and business organizations and construction firms to prefabricate all-inclusive ADU packages that would relieve sone of the cost burden on homeowners.
“Creating ADUs is a great way to add more housing opportunities in existing single-family neighborhoods,” Housing Trust CEO Kevin Zwick said in a press release. “We’re happy to see San Jose continue to use innovative tools to address housing in the region and invite everyone from philanthropic and corporate partners to homeowners to become part of the solution.”
The proposal garnered support from council members Magdalena Carrasco, Sylvia Arenas and Pam Foley. The entire council is set to discuss the YIMBY idea as part of its budget hearings this Tuesday.
Purdue Pharma reaches tentative settlement in federal opiods lawsuit
Purdue Pharma reaches tentative settlement in federal opiods lawsuit and some state litigation
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September 11, 2019 at 10:50 a.m. PDT
Under terms of a plan that has been under discussion for months, the Sacklers would relinquish control of their company. Stamford, Conn.-based Purdue Pharma would declare bankruptcy and be resurrected as a trust whose main purpose would be to combat the opioid epidemic.
The deal is said to be worth $10 billion to $12 billion, including a $3 billion payment from the Sacklers’ personal funds. It also would include money from the sale of the family’s international drug conglomerate, Mundipharma, according to the documents and people close to the talks.
For many attorneys general, that proposal apparently was good enough. The main objection from states that opposed the plan was that the Sacklers were not contributing enough cash from their personal fortunes, built almost entirely on the sale of OxyContin and taken out of the company in recent years.
It was not clear Wednesday whether the Sacklers had agreed to increase their personal contribution to the settlement.
‘Superagers’ have memory and brain connectivity of twenty-somethings
Summary: Both the default mode network and salience network in superagers had stronger connectivity than typical older adults and similar connectivity as younger adults. Superagers performed similarly to young adults and better than typical older adults in recognition and episodic memory tasks.
Source: Mass General
Research published in the journal Cerebral Cortex has shown that stronger functional connectivity—that is, communication among neurons in various networks of the brain—is linked to youthful memory in older adults. Those with superior memories—called superagers—have the strongest connectivity.
The work is the second in a series of three studies undertaken to unlock the secret of something researchers already knew: that some adults in their 80s and 90s function cognitively as well as or better than much younger people.
The first study showed that when compared with typical older adults, the brains of superagers are larger in certain areas that are important for processes that contribute to memory, including learning, storing, and retrieving information. But brain regions are not isolated islands; they form networks that “talk” to one another to allow for complex behaviors.
“This communication between brain regions is disrupted during normal aging,” said Alexandra Touroutoglou, Ph.D., an investigator in the MGH Department of Neurologyand the Athinoula A. Martinos Center for Biomedical Imaging. “Superagers show not just youthful brain structure, but youthful connectivity as well.”
The current study looked at superagers, typical adults from 60 to 80 years old, and young adults 18 to 35. It used functional magnetic resonance imaging (fMRI) to examine the synchronization of brain waves in the default mode network (DMN) and salience network (SN) of participants in a resting state.
“These networks ebb and flow, or oscillate, whether you’re in a resting state or engaged in a task,” said Bradford C. Dickerson, MD, director of MGH’sFrontotemporal Disorders Unit. “Our prediction was that typical older adults would have less synchronization in these brain waves—less efficient networks—but that superagers would have networks as efficient as the young adults. And that’s what we found.”
The research team’s next study will analyze fMRI data from brains engaged in memory and other cognitive tasks. It is hoped that taken together, the studies will “provide basics for future researchers to develop biomarkers of successful aging,” said Touroutoglou, who is also an instructor in neurology at Harvard Medical School, noting that one of the mysteries scientists hope to tease out is whether superagers start off with “bigger and better” brain structure and communication than other people or if they are somehow more resilient to the declines of normal aging. Future research may then measure the effects of genetics as well as exercise, diet, social connectedness, and other lifestyle factors that have been shown to affect resilience in older adults.
“We hope to identify things we can prescribe for people that would help them be more like a superager,” said Dickerson, who is also an associate professor of neurology at Harvard Medical School. “It’s not as likely to be a pill as more likely to be recommendations for lifestyle, diet, and exercise. That’s one of the long-term goals of this study—to try to help people become superagers if they want to.”
ABOUT THIS NEUROSCIENCE RESEARCH ARTICLE
Source:
Mass General
Media Contacts:
Terri Janos – Mass General
Image Source:
The image is in the public domain.
Original Research: Closed access
“Stronger Functional Connectivity in the Default Mode and Salience Networks Is Associated With Youthful Memory in Superaging “. Jiahe Zhang, Joseph M Andreano, Bradford C Dickerson, Alexandra Touroutoglou, Lisa Feldman Barrett.
Cerebral Cortex doi:10.1093/cercor/bhz071
Abstract
Stronger Functional Connectivity in the Default Mode and Salience Networks Is Associated With Youthful Memory in Superaging
“Superagers” are older adults who, despite their advanced age, maintain youthful memory. Previous morphometry studies revealed multiple default mode network (DMN) and salience network (SN) regions whose cortical thickness is greater in superagers and correlates with memory performance. In this study, we examined the intrinsic functional connectivity within DMN and SN in 41 young (24.5 ± 3.6 years old) and 40 older adults (66.9 ± 5.5 years old). Superaging was defined as youthful performance on a memory recall task, the California Verbal Learning Test (CVLT). Participants underwent a resting-state functional magnetic resonance imaging (fMRI) scan and performed a separate visual–verbal recognition memory task. As predicted, within both DMN and SN, superagers had stronger connectivity compared with typical older adults and similar connectivity compared with young adults. Superagers also performed similarly to young adults and better than typical older adults on the recognition task, demonstrating youthful episodic memory that generalized across memory tasks. Stronger connectivity within each network independently predicted better performance on both the CVLT and recognition task in older adults. Variation in intrinsic connectivity explained unique variance in memory performance, above and beyond youthful neuroanatomy. These results extend our understanding of the neural basis of superaging as a model of successful aging.
Top health hacks 9-9-2019
“Eggplant and apple cider vinegar for skin cancer”
About Motherhealth bay area caregivers, free cancer ebook
Frequency of the human body, whole foods, canned foods
Do not need to work with debt settlement companies, call Chase 866-3510182
Iron and sweet wormwood herb kill breast cancer cells in 16hrs
Philippines Coconut Wine -Tuba
Foods to eat and avoid when you have Gout and leg pains
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Cash flow analysis worksheet template
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Root Canal and Implants by Dr Mercola
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Skin cancer stories
Eggplant and apple cider vinegar for skin cancer
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Signs of the preactive and active phase of dying, medications for terminally ill
Affordable senior housing in the bay area
Nitric Oxide Dump Exercise with nose breathing to lower blood pressure and thin blood
IHSS Santa Clara County pays caregivers $13 per hour to help you care for your aging parents
Pre-clinical planning and eval of medical devices
NAC, activated charcoal , sleep and parasites
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Daily Kos Recommended
Hyaluronic Acid rich foods to prevent epilepsy or seizures
Dr Mercola on Knee Osteoarthritis
Stem cell injections for osteoarthritis
Anti-aging and Parkinson/Alzheimer’s prevention: Enzymes and apple cider vinegar
Go to irs.gov and use there programs to negotiate for a payment plan
Pineapple, celery, carrots and Arthritis
The level of your hunger could determine your bone structure
Tryptophan – Niacin – NAD = Anti-aging
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How to remove fraudulent bills in your checking account?
How to have no monthly car payments?
Save for half the amount of a use car, less than $10k price with low mileage. Use the savings for 50% of the down payment and do a loan for the balance which you will budget to pay within 4 months. You then will never have to pay monthly car payments for the next 3 to 6 years. Live simply and within your means.
