What I Learned About Eye Floaters


What I Learned About Eye Floaters

an-eye-chart-to-test-visionI am a retired Certified Ophthalmic Medical Technologist, so let me begin by giving you my “professional” opinion. Floaters were a common complaint I heard about, and this is what I used to tell patients. Some of it is actually good advice!

The eye has two compartments filled with different gel-like substances. At the front of the eye is aqueous, but the bulk of the eye is filled with vitreous, which is water and collagen, and it can break down as we age. When it breaks down it forms little bubbles and those bubbles cast a shadow on the retina, generating the appearance of dark spots floating around in the visual field. These spots are harmless and may fade, but will likely remain visible for the rest of your life. People tend to get used to them and they become less annoying. If it becomes so severe that it impairs the vision, there is a surgical technique to suck the vitreous out and replace it, but it is quite risky and expensive.

Read more: Natural Options for Treating Eye Floaters

Why You Should Not Ignore Your Eye Floaters

Floaters can be a very serious warning sign of a potentially blinding condition. If your floaters seem to form a web or a curtain, or if they are accompanied by flashes of light you need to get to an ophthalmologist IMMEDIATELY. The vitreous is adhered to the retina, and when the vitreous degrades it can pull the retina off. If you catch it early, the retina can be “tacked” back on with a laser quickly and almost painlessly. If it goes on too long, the retina requires major surgery and if it goes too far you loose the vision in that eye. There is no saving it once the retina peels off your macula (the central “sweet spot” of vision is processed there). If there is any pain, you ought to be checked out by a doctor. Floaters should never be painful.

Officially, and essentially, floaters themselves are harmless and there is no treatment, so get used to them. This is what I learned, and repeated many times. Then one day I got floaters and suddenly the advice I had given for decades was not adequate. I did go see an ophthalmologist. The risk of blindness is serious and I was not playing around. His assistant was a former co-worker who dutifully gave me the same advice I had been giving when I was a “professional” in the field. I was told my vitreous had not detached, but it probably would within the month based on my symptoms. Vitreous detachment is not as serious as retinal detachment, but can lead to a retinal detachment.

Read more: The Medicine Your Grandmother Used

My Home Cure for Eye Floaters

I am not a doctor, but I am more than willing to play one on myself, and so I applied my general knowledge of alternative healing to the problem. I figured I had two problems that needed to be addressed: 1) inadequate blood supply to my eye, and 2) collagen breakdown.

Improving the blood supply to my eye was a goal based on the simple premise that when there is decreased health in the tissues, it will boil down to the cells not getting enough oxygen. I went with citrus bioflavanoids, a supplement well known to strengthen blood vessels. To help build up collagen, I began taking glucosamine and chondroitan. Within 2 weeks, I cancelled my follow up appointment, the floaters had dissolved.

If you do not have access to these supplements, try a diet rich in the same nutrients. The white part of orange peels is an excellent source of bioflavanoids. But there are tastier sources like red bell peppers, garlic, and strawberries. Glucosamine and chondroitan are a bit trickier to get large doses of in your diet. I would make a good bone sauce, and a nice jambalaya where you eat the seafood – shells and all, and eat lots of them both.

Connie’s comments:

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Tips: Drink fresh juice of carrots, orange, pineapple and add turmeric powder. Deep breathing IN thru noise and out thru mouth. Sleep early and adequately. Detox parasites and fungus in the body. Get Vitamin D from the sun. Strengthen collagen and blood vessels with ginger, soft boiled eggs and Vitamin C. And more. Email motherhealth@gmail.com



Protect your eyes and kidneys with anti-oxidants

Moderate exercise combined with dietary vitamins C and E counteracts oxidative stress in the kidney and lens of streptozotocin-induced diabetic-rat.


Oxidative stress has a key role in the pathogenesis of diabetes-induced cataract formation and nephropathy.

Daily moderate exercise and vitamins C and E (VCE) supplementation can be beneficial to diabetes due to reducing blood glucose and free radical production.

The aim of this study was to analyze the effect of moderate exercise with vitamin VCE on lipid peroxidation (LP) and antioxidative systems in the kidneys and lens of streptozotocin-induced diabetic rats. Forty female Wistar rats were used.

They were randomly divided into four groups. The first and second groups were used as control and diabetic groups. The third group was the diabetic-exercise group. VCE-supplemented feed was given to diabetic-exercise rats constituting the fourth group. Animals in the exercised groups were moderately exercised daily on a treadmill for three weeks (five days a week).

Diabetes was induced on day zero of exercise. Body weights in the four groups were recorded weekly. Lens and kidney samples were taken from all animals on day 20.

Glutathione peroxidase (GSH-Px), reduced glutathione (GSH), vitamin E, and beta-carotene levels in kidney and lens, albumin in plasma, and body weight were significantly lower in the diabetic group than in the control group, whereas there was a significant increase in LP of kidney and lens as well as plasma glucose, urea, and creatinine levels in the diabetic group.

The decrease in antioxidant enzymes, vitamins, and albumin and the increase in LP and glucose levels in diabetic rats were significantly improved with exercise and VCE supplementation. In the diabetic animals, the decreased beta-carotene and vitamins A levels in kidney did not improve through exercise only, although their levels were increased by exercise plus VCE supplementation.

In conclusion, these data demonstrate that lipid peroxidation increases in the lens and kidney of diabetic animals and this could be due to decreases in antioxidant vitamins and enzymes.

However, dietary VCE with moderate exercise may strengthen the antioxidant defense system through the reduction of ROS and blood glucose levels.

The VCE supplementations with exercise may play a role in preventing the development of diabetic nephropathy and cataract formation in diabetic animals.

Ditch any obsession with “anti-aging.” It actually accelerates aging! People with negative feelings about their age die 7.5 years sooner than people who embrace their age, found one study.

Effects of antioxidant supplementation on insulin sensitivity, endothelial adhesion molecules, and oxidative stress in normal-weight and overweight young adults.

Vincent HK1Bourguignon CMWeltman ALVincent KRBarrett EInnes KETaylor AG.

Author information


The objective of the study was to determine whether short-term antioxidant (AOX) supplementation affects insulin sensitivity, endothelial adhesion molecule levels, and oxidative stress in overweight young adults.

A randomized, double-blind, controlled study tested the effects of AOXs on measures of insulin sensitivity (homeostasis model assessment [HOMA]) and quantitative insulin sensitivity check index), endothelial adhesion molecules (soluble intercellular adhesion molecule-1, vascular adhesion molecule, and endothelial-leukocyte adhesion molecule-1), adiponectin, and oxidative stress (lipid hydroperoxides) in overweight and normal-weight individuals (N = 48, 18-30 years).

Participants received either AOX (vitamin E, 800 IU; vitamin C, 500 mg; beta-carotene, 10 mg) or placebo for 8 weeks. The HOMA values were initially higher in the overweight subjects and were lowered with AOX by week 8 (15% reduction, P = .02). Adiponectin increased in both AOX groups.

Soluble intercellular adhesion molecule-1 and endothelial-leukocyte adhesion molecule-1 decreased in overweight AOX-treated groups by 6% and 13%, respectively (P < .05). Plasma lipid hydroperoxides were reduced by 0.31 and 0.70 nmol/mL in the normal-weight and overweight AOX-treated groups, respectively, by week 8 (P < .05).

Antioxidant supplementation moderately lowers HOMA and endothelial adhesion molecule levels in overweight young adults.

A potential mechanism to explain this finding is the reduction in oxidative stress by AOX.

Long-term studies are needed to determine whether AOXs are effective in suppressing diabetes or vascular activation over time.

Antioxidant supplementation lowers exercise-induced oxidative stress in young overweight adults.

Vincent HK1Bourguignon CMVincent KRWeltman ALBryant MTaylor AG.

Author information



To determine whether antioxidant (AOX) supplementation attenuates post-exercise oxidative stress and contributors to oxidative stress (inflammation, blood lipids) in overweight young adults.


This was a randomized, double-blind, controlled study. Overweight (BMI, 33.2 +/- 1.9 kg/m(2)) and comparative normal-weight (BMI, 21.9 +/- 0.5 kg/m(2)) adults 18 to 30 years old (total N = 48) were enrolled. Participants received either daily antioxidant (AOX) treatment (800 IU of vitamin E, 500 mg of vitamin C, 10 mg of beta-carotene) or placebo (PL) for 8 weeks for a total of four groups. All participants completed a standardized 30-minute cycle exercise bout at baseline and 8 weeks. Exercise-induced changes in lipid hydroperoxide (DeltaPEROX), C-reactive protein (DeltaCRP), interleukin-6 (DeltaIL-6), cholesterol subfractions, triglycerides, total AOX status (DeltaTAS), and adiponectin were assessed.


Exercise-induced DeltaPEROX was lower in the overweight-AOX group (0.09 nM/kg per min) compared with PL-treated overweight and normal-weight groups (0.98, 0.53 nM/kg per min) by 8 weeks (p < 0.05). Adiponectin was increased in both overweight and normal-weight AOX groups (22.1% vs. 3.1%; p < 0.05) but reduced in PL groups. DeltaIL-6, Deltatotal cholesterol, and Deltalow-density lipoprotein-cholesterol concentrations during exercise were lower in the AOX-treated groups compared with PL groups (all p < 0.05). After controlling for BMI, the Deltatotal cholesterol, Deltalow-density lipoprotein-cholesterol, Deltaadiponectin, and DeltaTAS explained 59.1% of the variance of the regression model of the DeltaPEROX by 8 weeks (total model R(2) = 0.600; p = 0.015).


AOX lowers exercise-induced oxidative stress in overweight adults. Inflammatory and lipid markers may also be attenuated with AOX. Further studies are needed to determine whether AOX may be used in cardiovascular disease prevention in the overweight population.


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