Taking care of my senior clients in the bay area have  taught me a lot of lessons. One client of mine had been giving her 90 yr old husband suffering from gout with a combo of Angelica (dried), Astralagus (dried) and Alove vera (fresh added in drinks). After many months, her hubby is now growing hair.

The immune boosting herbs, Angelica and Astralagus, reduce sinility or reverses the aging process, enhances the immune system and promote Bone Marrow Stromal Cell growth. Aloe vera helps in maintaining the alkalinity of the blood/body.

The following are  three scientific studies about these herbs:

1. Angelica and Astragalus, Chinese herbal medicine, are used as immunostimulants for enriching the blood, anti-tumor and antioxidation effects. However, the synergistic mechanisms of antioxidant effect have long been the object of intensive study. We investigated the deferring senile effect of AAP on aging mice by determination of the contents of monoamine oxidase (MAO) in brain, hydroxyproline (Hyp) in skin, hydrogen peroxide (H2O2) in serum, glutathione peroxidase (GSH-PX) and xanthine oxidase (XOD) in liver tissue and immunity organs index.
   The obtained results showed that AAP decreased significantly the content of MAO and H2O2, and enhanced the concentrations of Hyp, GSH-PX and XOD in vivo. No significant differences were found when compared with those of vitamin E. Additionally, AAP could increase immunity organs index. These results demonstrated that AAP had significant deferring senile effect on the aging mice. These changes may be the important mechanisms to prevent mice from aging [Human Health and Biomedical Engineering (HHBE), 2011 International Conference].
2. Astragalus with Angelica or used separately could promote BMSC proliferation. The mechanism might induce the VEGF protein expression in BMSC. And the independent use of Angelica has no above effect [PubMed].
3. Combined extract of Astragalus membranaceus and Angelica sinensis (A&A) is a traditional antifibrotic agent in China. The present investigation aimed to determine whether an ACEi (Enalapril) and A&A together have a better antifibrotic effect in unilateral ureteral obstruction (UUO) than monotherapy with either agent. Transforming growth factor-beta1, fibroblast activation, collagen deposition, macrophage accumulation and tubular cell apoptosis were all decreased.
   The combination of the two drugs was significantly more effective than Enalapril alone in reducing tumor necrosis factor-alpha, collagen accumulation, activation of fibroblasts, and tubular cell apoptosis. In conclusion, Enalapril with A&A significantly decreased tubulointerstitial fibrosis to a greater extent than treatment with Enalapril alone.