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Metal toxins , mercury dental amalgams, Alzheimers, brain detox

I am caring for a 92 yr old with Dementia and she has metal dental fillings.  I started her with whole foods, exercise, sunshine and loving care with massage.  At night she has anxiety disorder.  I wish her dental metal fillings are removed as it can be toxic affecting her memory cells.  I buy cilantro, garlic, onions and healthy drinks of lemon and orange juice for her.

Soon I will start her with quality supplementation at:

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mercury amalgam

Association between dental amalgam fillings and Alzheimer’s disease: a population-based cross-sectional study in Taiwan

The potential effects of amalgam fillings on the development of Alzheimer’s disease (AD) are not well understood. The aim of the study was to evaluate the association between dental amalgam fillings and Alzheimer’s disease in Taiwanese population aged 65 and older.

Methods

Data were retrieved from the Longitudinal Health Insurance Database (LHID 2005 and 2010). The study enrolled 1,943,702 beneficiaries from the LHID database. After excluding death cases and individuals aged 65 and under, 207,587 enrollees were finally involved in the study. Dental amalgam fillings are coded as 89001C, 89002C, 89003C, 89101C, 89102C, or 89103C in the national health insurance research database (NHIRD). Alzheimer’s disease was diagnosed using the International Classification of Diseases, Ninth Revision, Clinical Modification (ICD-9-CM) codes 331.0.

Results

Individuals exposed to amalgam fillings had higher risk of Alzheimer’s disease (odds ratio, OR = 1.105, 95 % confidence interval, CI = 1.025-1.190) than their non-exposed counterparts. Further analysis showed that the odds ratio of Ahlzheimer’s disease was 1.07 (95 % CI = 0.962-1.196) in men and 1.132 (95 % CI = 1.022-1.254) in women.

Conclusions

Women who were exposed to amalgam fillings were 1.132 times more likely to have Alzheimer’s disease than were their non-exposed counterparts.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4642684/

 

Numerous studies have also demonstrated that the mercury exposure or concentration increases in the following tissues and situations

  • Due to chewing, brushing, and/or bruxism
  •  In exhaled or intra-oral air of persons with amalgam fillings
  •  In saliva of persons with amalgam fillings
  •  In blood of persons with amalgam fillings
  •  In various organs and tissues of amalgam bearers, including the kidney, liver, pituitary gland, thyroid, and brain or parts thereof
  •  In feces of amalgam bearers
  •  In amniotic fluid, cord blood, placenta, and various fetal tissues including liver, kidney and brain, in association with maternal amalgam load
  •  In colostrum and breast milk in association with maternal amalgam load

Scientific evidence confirms that in most individuals with dental mercury amalgam fillings, mercury exposure exceeds the Reference Exposure Level (REL).

[REL is a term used to denote the exposure level defined by national and international regulatory agencies at which there is an expectation of no negative health outcomes within the population.]
Also, reports from the World Health Organization (WHO) and Canada’s federal department of health (Health Canada) conclude that mercury vapor from dental amalgam is the greatest source of human exposure to mercury in non-industrial settings.

Additionally, in research published in 2011, Dr. G. Mark Richardson reported that more than 67 million Americans aged two years and older exceed the intake of mercury vapor considered “safe” by the U.S. EPA due to the presence of dental mercury amalgam fillings, whereas over 122 million Americans exceed the intake of mercury vapor considered “safe” by the California.

Click to access IAOMT-Position-Statement-Update-2016-6.16.16.pdf

Hospitals are housing the homeless: Will it help?

Hospitals are housing the homeless: Will it help?

Hospitals in Sacramento and around the country are taking steps to help homeless people find housing. Doing so, they say, will limit unnecessary ER visits and reduce wasteful healthcare spending. It also helps nonprofits such as San Francisco-based Dignity Health, Orlando-based Florida Hospital and Providence Health & Services in Portland, Ore., meet their community service obligations in exchange for tax breaks.

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Looking for investors or those who wants to donate their lands to Green Research Instititute 501c3. 1708 Hallmark lane san jose ca 95124. Connie Dello Buono. 408-854-1883 motherhealth@gmail.com

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Lower Brain Glucose Levels in Brains of People With Obesity and Type 2 Diabetes

Lower Brain Glucose Levels in Brains of People With Obesity and Type 2 Diabetes

Summary: Yale researchers report people with type 2 diabetes and obesity have reduced levels of glucose in their brains. The finding may help explain why obese people and those with T2DM have increased risk of eating disorders and Alzheimer’s disease.

Source: Yale.

Glucose levels are reduced in the brains of individuals with obesity and type 2 diabetes compared to lean individuals, according to a new Yale study. The finding might explain disordered eating behavior — and even a higher risk of Alzheimer’s disease — among obese and diabetic individuals, the researchers said.

The study was published Oct. 19 in JCI Insight.

Both obesity and type 2 diabetes are linked to decreased metabolism in the brain. This hypometabolism is also associated with Alzheimer’s disease, but researchers have not pinpointed why. To examine the mechanism, the Yale team studied brain glucose levels in three different groups of adults: individuals who are lean and healthy, and those with either obesity or poorly controlled type 2 diabetes.

After fasting overnight, the study participants received intravenous infusions of glucose for two hours. During the infusions, the researchers used a brain scanning technique — magnetic resonance spectroscopy — to measure levels of glucose in the brain.

While blood glucose levels among the participants were similar, the researchers detected significant differences in brain glucose. Among the obese and diabetic participants, “we found decreased or blunted entry of glucose into the brain,” said first author and assistant professor of medicine Janice Hwang, M.D.

That blunting could be one mechanism that undermines the ability of the brain to sense glucose, she noted.

Image shows a brain model.

The researchers also rated participants’ hunger, satisfaction, and fullness before and after the infusions. “The lean people who had more glucose entry into the brain also felt more full, even though they hadn’t eaten overnight,” she said.

Hwang explained further: “Glucose is the most primitive signal to the brain that you’ve eaten. Could it be that obese individuals are not getting sugar into the brain, and not sensing it; thus the feedback loop to stop eating could also be blunted?”

The study points to the importance of sugar transport from the blood into the brain as both a target for further research and possible pharmacological intervention in people with obesity and type 2 diabetes, the researchers noted.

ABOUT THIS NEUROSCIENCE RESEARCH ARTICLE

Other study authors are Lihong Jiang, Muhammad Hamza, Elizabeth Sanchez Rangel, Feng Dai, Renata Belfort-DeAguiar, Lisa Parikh, Brian B. Koo, Douglas L. Rothman, Graeme Mason, and Robert S. Sherwin.

Funding: This study was supported in part by grants from the National Institutes of Health, and the Yale Center for Clinical Investigation, supported by the Clinical and Translational Science Award, the Endocrine Fellows Foundation, and the American Diabetes Association. Hwang reports research support from Pfizer and Regeneron.

Source: Ziba Kashef – Yale
Publisher: Content organized by NeuroscienceNews.com.
Image Source: NeuroscienceNews.com image is adapted from the Yale news release.
Original Research: Full open access research for “Blunted rise in brain glucose levels during hyperglycemia in adults with obesity and T2DM” by Janice J. Hwang, Lihong Jiang, Muhammad Hamza, Elizabeth Sanchez Rangel, Feng Dai, Renata Belfort-DeAguiar, Lisa Parikh, Brian B. Koo, Douglas L. Rothman, Graeme Mason, Robert S. Sherwin in JCI Insight. Published online October 19 2017 doi:10.1172/jci.insight.95913

CITE THIS NEUROSCIENCENEWS.COM ARTICLE
Yale “Lower Brain Glucose Levels in Brains of People With Obesity and Type 2 Diabetes.” NeuroscienceNews. NeuroscienceNews, 19 October 2017.
<http://neurosciencenews.com/brain-glucose-obesity-diabetes-7771/&gt;.

Abstract

Blunted rise in brain glucose levels during hyperglycemia in adults with obesity and T2DM

n rodent models, obesity and hyperglycemia alter cerebral glucose metabolism and glucose transport into the brain, resulting in disordered cerebral function as well as inappropriate responses to homeostatic and hedonic inputs. Whether similar findings are seen in the human brain remains unclear. In this study, 25 participants (9 healthy participants; 10 obese nondiabetic participants; and 6 poorly controlled, insulin- and metformin-treated type 2 diabetes mellitus (T2DM) participants) underwent 1H magnetic resonance spectroscopy scanning in the occipital lobe to measure the change in intracerebral glucose levels during a 2-hour hyperglycemic clamp (glucose ~220 mg/dl). The change in intracerebral glucose was significantly different across groups after controlling for age and sex, despite similar plasma glucose levels at baseline and during hyperglycemia. Compared with lean participants, brain glucose increments were lower in participants with obesity and T2DM. Furthermore, the change in brain glucose correlated inversely with plasma free fatty acid (FFA) levels during hyperglycemia. These data suggest that obesity and poorly controlled T2DM progressively diminish brain glucose responses to hyperglycemia, which has important implications for understanding not only the altered feeding behavior, but also the adverse neurocognitive consequences associated with obesity and T2DM.

“Blunted rise in brain glucose levels during hyperglycemia in adults with obesity and T2DM” by Janice J. Hwang, Lihong Jiang, Muhammad Hamza, Elizabeth Sanchez Rangel, Feng Dai, Renata Belfort-DeAguiar, Lisa Parikh, Brian B. Koo, Douglas L. Rothman, Graeme Mason, Robert S. Sherwin in JCI Insight. Published online October 19 2017 doi:10.1172/jci.insight.95913

Antioxidants and insulin sensitivity

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Researchers have shown that red wine and tea may both hold promise for regulating the blood sugar of people with type 2 diabetes.

Levels of blood glucose rise sharply in patients with type 2 diabetes immediately following a meal. Red wine and tea contain antioxidants that help to slow the passage of glucose through the small intestine and into the bloodstream, which can prevent the blood sugar spike.

Keeping blood sugar levels normal is one of the key challenges of managing diabetes; doing so can help prevent the disease from contributing to heart disease and high blood pressure as well as damaging the eyes, kidneys, nerves and blood vessels.

In the study, researchers tested how well wine and tea could inhibit the activity of a target enzyme called alpha-glucosidase, which is responsible for triggering the absorption of glucose by the small intestine.

Red wine came out on top, as it was able to inhibit the enzyme by nearly 100 percent, compared to white wine at 20 percent.

Out of the four types of tea tested — black, oolong, white and green — black tea was most effective, followed by white tea and oolong tea.

Dr. Mercola’s Comments:
Red wine and tea continue to make headlines as some of the most promising “functional foods” of the 21st century. But it takes a little bit of digging to get to the bottom of what’s really healthy, and not so healthy, about these two beverages.

Should You Drink Red Wine?

No doubt about it, red wine contains lots of antioxidants, namely polyphenols, which are known to fight free radicals and reduce your risk of a number of degenerative conditions from cancer and heart disease to neurodegenerative diseases.

And resveratrol, perhaps the most talked about antioxidant in red wine, may even extend your lifespan.

So should you have a glass now and then?

In my opinion, no. This is largely because, despite the other healthy properties in red wine, the alcohol itself is actually a neurotoxin, which means it can poison your brain. Additionally, it has the strong potential to seriously disrupt your delicate hormone balance. This may be why if you drink heavy amounts of beer and spirits you may double your risk of developing colorectal tumors.

You also need to be aware that consuming large amounts of wine will increase insulin levels and eventually have a negative impact on your health. This is especially important for people who already show signs of insulin resistance, such as high blood pressure, extra weight, high cholesterol, and, yes, diabetes.

This is contradictory to the study above, which found that wine actually benefits blood sugar. Well, keep in mind that this study only looked at wine’s ability to inhibit the activity of a single target enzyme, and not how it would impact your entire system.

Having said that, it is important to understand some important facts about resveratrol. Because this antioxidant is soluble in alcohol, you will get far more absorption if you consume it in an alcohol base as opposed to swallowing it from a pill. So while there are clearly distinct and negative consequences to consuming alcohol, these are partially compensated for by its ability to increase the absorption of resveratrol into your blood where it performs its magic.

Keep in mind, though, that if you decide to drink red wine, you need to check on the growing conditions of the grapes and how the wine is made. If the wine isn’t made with organic grapes, it may contain no resveratrol at all.

Should You Drink Tea?

Like wine, tea is packed with antioxidants that are great for your health. Research has indicated that tea could have beneficial effects including:

  • Improved mental alertness
  • Lower blood cholesterol and triglyceride levels
  • Reduced blood pressure
  • Lower risk of breast, colon, lung, ovarian and prostate cancers
  • Protection again type 2 diabetes

And one component of tea, epigallocatechin gallate (EGCG), could also help prevent psoriasis, prostate cancer and colon tumors. Several studies have also found that EGCG can improve exercise performance, increase fat oxidation and prevent obesity, as it’s known to have a regulatory effect on fat metabolism.

So should you make a cup of tea, or a few of them, part of your day?

Well, next to pure water, high-quality tea is one of the most nutritious beverages you can consume. Personally, I’ve begun to include matcha green tea from 100 percent, quality tea leaves as a regular part of my diet.

How Else Can You Regulate Your Blood Sugar?

Please don’t misconstrue — there are many ways to improve your health and your blood sugar levels that don’t involve drinking anything. Among the most powerful:

1. Find out your nutritional type. Nearly all type 2 diabetics need to swap out their grains and sugars for other foods, however, some of you will benefit from using protein for the substitution, while others will benefit from using more vegetable-only carbohydrates. Which one is determined by your nutritional type.

2. Exercise. Regular physical activity helps to stabilize your blood sugar and make your cells more responsive to insulin. So get moving!

3. Use cinnamon. If you enjoy this spice, feel free to sprinkle it on your food generously — it’s known to help control blood sugar levels. Though this doesn’t address the underlying causes of type 2 diabetes like the first two tips do, it’s still an inexpensive, and safe, tool to add to your collection.

Connie’s comments:

Greens, ginger, garlic, onions, massage, de-stressing, exercise, adequate sleep are all factors that can help regulate your sugar blood levels.

 

—————

Effects of antioxidant supplementation on insulin sensitivity, endothelial adhesion molecules, and oxidative stress in normal-weight and overweight young adults.

Abstract

The objective of the study was to determine whether short-term antioxidant (AOX) supplementation affects insulin sensitivity, endothelial adhesion molecule levels, and oxidative stress in overweight young adults.

A randomized, double-blind, controlled study tested the effects of AOXs on measures of insulin sensitivity (homeostasis model assessment [HOMA]) and quantitative insulin sensitivity check index), endothelial adhesion molecules (soluble intercellular adhesion molecule-1, vascular adhesion molecule, and endothelial-leukocyte adhesion molecule-1), adiponectin, and oxidative stress (lipid hydroperoxides) in overweight and normal-weight individuals (N = 48, 18-30 years).

Participants received either AOX (vitamin E, 800 IU; vitamin C, 500 mg; beta-carotene, 10 mg) or placebo for 8 weeks. The HOMA values were initially higher in the overweight subjects and were lowered with AOX by week 8 (15% reduction, P = .02).

Adiponectin increased in both AOX groups. Soluble intercellular adhesion molecule-1 and endothelial-leukocyte adhesion molecule-1 decreased in overweight AOX-treated groups by 6% and 13%, respectively (P < .05).

Plasma lipid hydroperoxides were reduced by 0.31 and 0.70 nmol/mL in the normal-weight and overweight AOX-treated groups, respectively, by week 8 (P < .05). Antioxidant supplementation moderately lowers HOMA and endothelial adhesion molecule levels in overweight young adults.

A potential mechanism to explain this finding is the reduction in oxidative stress by AOX.

PMID:

 

19154960

 

PMCID:

 

PMC3325609

 

DOI:

 

10.1016/j.metabol.2008.09.022

How to choose the right eye nutrients , AREDS – Age-Related Eye Disease Studies

Eye supplements based on AREDS 2 (Age-Related Eye Disease Study) can be found at:

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Email motherhealth@gmail.com for nutrition protocol and nutrition testers integrated in doctor’s practice and health care pros. We are looking for distributors and reps in 54 countries.

eye formula

 

What the Age-Related Eye Disease Studies Mean for You

Researchers with the Age-Related Eye Disease Study (AREDS) reported in 2001 that a nutritional supplement called the AREDS formulation can reduce the risk of developing advanced age-related macular degeneration (AMD). The original AREDS formulation contains vitamin C, vitamin E, beta-carotene, zinc and copper.

In 2006, the same research group, which is based at NIH’s National Eye Institute, began a second study called AREDS2 to determine if they could improve the AREDS formulation. They tested…

  • Adding the antioxidants lutein and zeaxanthin
  • Adding omega-3 fatty acids
  • Removing beta-carotene
  • Lowering the dose of zinc

This page provides information about the results and implications of AREDS2. If you are at risk for advanced AMD or have a family member who is at risk, the questions and answers below may help you discuss using AREDS and related nutritional supplements with a health care professional.

What is the original AREDS formulation?

  • 500 milligrams (mg) of vitamin C
  • 400 international units of vitamin E
  • 15 mg beta-carotene
  • 80 mg zinc as zinc oxide
  • 2 mg copper as cupric oxide

What modifications were tested in AREDS2?

  • 10 mg lutein and 2 mg zeaxanthin
  • 1000 mg of omega-3 fatty acids (350 mg DHA and 650 mg EPA)
  • No beta-carotene
  • 25 mg zinc

Why change the formulation?

Why add lutein/zeaxanthin and omega-3 fatty acids? Previous studies had found that dietary intake of lutein/zeaxanthin and omega-3 fatty acids is associated with a lower risk of developing advanced AMD.

Why eliminate beta-carotene? During the AREDS trial, two large trials funded by the National Cancer Institute found that beta-carotene may increase lung cancer risk among people who smoke. Lutein and zeaxanthin are in the same family of nutrients as beta-carotene and are believed to have important functions in the retina. Therefore, the researchers theorized that lutein/zeaxanthin might be a safer and possibly more effective alternative than beta-carotene.

Why reduce zinc? Although zinc was found to be an essential component of the AREDS formulation in the original trial, some nutritional experts recommended a lower dose.

What formulation should I take?

Consult your doctor or eye care professional about which supplement, if any, is right for you. The ingredients based on AREDS and AREDS2 research are:

  • 500 milligrams (mg) of vitamin C
  • 400 international units of vitamin E
  • 80 mg zinc as zinc oxide
  • 2 mg copper as cupric oxide
  • 10 mg lutein and 2 mg zeaxanthin

What are lutein, zeaxanthin and beta-carotene?

Lutein, zeaxanthin, and beta-carotene belong to a family of nutrients known as carotenoids. Carotenoids are made by plants and are especially enriched in green leafy vegetables. They can be stored in animal tissues and are found at relatively low levels in animal food products. In the body, beta-carotene is used to make Vitamin A, which is required by the retina to detect light and convert it into electrical signals. Beta-carotene itself is not found in the eye. In contrast, lutein and zeaxanthin are found in the retina and lens, where they may act as natural antioxidants and help absorb damaging, high-energy blue and ultraviolet light.

What are omega-3 fatty acids?

Omega-3 fatty acids are made by marine algae and enriched in fish oils; they are believed to be responsible for the health benefits associated with regularly eating fish, including lower rates of cardiovascular disease. The AREDS2 study focused on the omega-3 fatty acids DHA and its precursor EPA. DHA is needed for the integrity of the retinal cells, and has been shown to promote retinal development and repair in prior studies.

What were the effects of changing the original AREDS formulation?

In the first AREDS trial, taking the original formulation reduced the risk of advanced AMD by about 25 percent over a five-year period. In the AREDS2 trial, adding DHA/EPA or lutein/zeaxanthin to the original formulation (containing beta-carotene) had no additional overall effect on the risk of advanced AMD. However, trial participants who took AREDS containing lutein/zeaxanthin and no beta-carotene had a slight reduction in risk of advanced AMD, compared with those who took AREDS with beta-carotene. Also, for participants with very low levels of lutein/zeaxanthin in their diet, adding these supplements to the AREDS formulation helped lower their risk of advanced AMD. Finally, former smokers who took AREDS with beta-carotene had a higher incidence of lung cancer. (Please see below for more details on the effects of lutein/zeaxanthin vs. beta-carotene.) The investigators found no significant changes in the effectiveness of the formulation when they removed beta-carotene or lowered zinc.

Who should consider taking a combination of antioxidants and zinc like those examined in AREDS and AREDS2?

People at high risk for developing advanced AMD should consider taking the antioxidant-zinc combinations examined in AREDS and AREDS2. These people are defined as having either:

  1. Intermediate AMD in one or both eyes. Intermediate AMD can be detected by an eye care professional, but usually involves little or no vision loss.
  2. Advanced AMD in one eye, but not the other eye. Advanced AMD involves either a breakdown of cells in the retina (called geographic atrophy or dry AMD), or the growth of abnormal blood vessels under the retina (called neovascular or wet AMD). Either of these forms of advanced AMD can cause vision loss.

Will taking an AREDS formulation prevent AMD?

There is no known treatment that can prevent the early stages of AMD. However, the AREDS formulations may delay progression of advanced AMD and help you keep your vision longer if you have intermediate AMD, or advanced AMD in one eye. The participants in the first AREDS trial have now been followed for 10 years, and the benefits of the AREDS formulation have persisted over this time.

Can I take a daily multivitamin if I am taking one of the AREDS formulations?

Yes. The AREDS formulation is not a substitute for a multivitamin. In the AREDS trial, two-thirds of the study participants took multivitamins along with the AREDS formulation. In AREDS2, almost nine of ten participants took multivitamins.

Can a daily multivitamin alone provide the same vision benefits as an AREDS formulation?

No. The vitamins and minerals tested in the AREDS and AREDS2 trials were provided in much higher doses than what is found in multivitamins. Also, it is important to remember that most of the trial participants took multivitamins. Taking an AREDS formulation clearly provided a benefit over and above multivitamins.

Can diet alone provide the same high levels of antioxidants and zinc as the AREDS formulations?

No. The high levels of vitamins and minerals are difficult to achieve from diet alone. However, previous studies have suggested that people who have diets rich in green, leafy vegetables—a good source of lutein/zeaxanthin—have a lower risk of developing AMD. In the AREDS2 trial, the people who seemed to benefit most from taking lutein/zeaxanthin were those who did not get much of these nutrients in their diet. Within this group, those who received lutein/zeaxanthin supplements had a 26 percent reduced risk of developing advanced AMD compared with those who did not receive the supplements.

What is the risk of lung cancer from taking beta-carotene?

In the AREDS2 trial, current smokers or those who had quit smoking less than a year before enrollment were excluded from receiving beta-carotene. Despite this precaution, lung cancers were observed in 2 percent of participants who took an AREDS formulation with beta-carotene, compared with 0.9 percent of participants who took AREDS without beta-carotene. Across both groups, about 91 percent of participants who developed lung cancer were former smokers.

How does lutein/zeaxanthin compare to beta-carotene?

Lutein/zeaxanthin has not been associated with increased cancer risk. Moreover, analysis from the AREDS2 trial suggests that it offers similar or better protective benefits against advanced AMD, compared with beta-carotene. In the trial, participants who took an AREDS formulation containing lutein/zeaxanthin (no beta-carotene) had an 18 percent lower risk of progressing to advanced AMD compared with those who took AREDS containing beta-carotene (no lutein/zeaxanthin).

Does the high-dose vitamin E in the AREDS formulations affect the risk of prostate cancer?

There have been conflicting data on the relationship between vitamin E and prostate cancer.

  • In 1994, the Alpha-Tocopherol, Beta Carotene (ATBC) trial found a 35 percent reduced risk of prostate cancer in men taking 50 mg of vitamin E daily for a follow-up of six years.
  • In 2009, the Physicians Health Study II (PHS II) found that 400 IU of vitamin E every other day for a follow-up of eight years had no effect on the incidence of prostate cancer.
  • In 2011, the Selenium and Vitamin E Cancer Prevention Trial (SELECT) found a 17 percent increase in the risk of prostate cancer among men taking 400 IU of vitamin E daily for a follow-up of seven years. That risk equates to 1-2 more prostate cancers per 1000 patients who took high-dose vitamin E for one year. For reasons that are unclear, men who took both vitamin E and selenium did not have an increased rate of prostate cancer.

In the AREDS trial, high-dose vitamin E had no effect on the risk of prostate cancer among male participants. The AREDS2 trial began in 2006 (before the SELECT trial was reported) and all study participants were offered an AREDS formulation containing vitamin E. A group of independent researchers monitoring the AREDS2 trial for safety noted no concerns about an increased risk prostate cancer. The final data from the study do not suggest a higher rate of prostate cancer among male participants than expected in an aging male population.

If you have concerns about vitamin E and prostate cancer, it is important to understand that many factors influence the risk of prostate cancer, including age, family history and race. Visit the National Cancer Institute web site for more information about prostate cancer risk factors(link is external), and talk to your health care provider about the possible risks and benefits from taking vitamin E supplements.

Are there any other side effects or risks from taking the AREDS formulations?

Many older Americans take prescription medications, and a considerable number use over-the-counter drugs, dietary supplements, and herbal medicines. High-dose supplemental nutrients can sometimes interfere with medications and compete with other vital nutrients for absorption into the body. Individuals who are considering taking an AREDS formulation should discuss this with their primary care doctors and/or eye care professionals.

Last Updated: July 2017

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