Home Care California | The Best Home Care Services

Home Care and Home Health: 4088541883

Natural communication breakdown in aging

A naturally produced compound rewinds aspects of age-related demise in mice. Researchers have discovered a cause of aging in mammals that may be reversible. The essence of this finding is a series of molecular events that enable communication inside cells between the nucleus and mitochondria. As communication breaks down, aging accelerates. By administering a moleculeContinue reading “Natural communication breakdown in aging”

Resveratrol and calorie restriction activates SIRT1 , anti-aging gene

When the SIRT1 gene is activated, it produces proteins that protect cells from inflammation and oxidative stress, two of the primary causes of premature aging and many degenerative diseases. Almost all organisms have a gene that is equivalent to SIRT1. The gene is a defense mechanism, activated by low calorie consumption, that gives cells anContinue reading “Resveratrol and calorie restriction activates SIRT1 , anti-aging gene”

THE LINK BETWEEN CIRCADIAN RHYTHMS AND AGING

MIT study finds that a gene associated with longevity also regulates the body’s circadian clock. Human sleeping and waking patterns are largely governed by an internal circadian clock that corresponds closely with the 24-hour cycle of light and darkness. This circadian clock also controls other body functions, such as metabolism and temperature regulation. Studies inContinue reading “THE LINK BETWEEN CIRCADIAN RHYTHMS AND AGING”

Sirt1 Regulates Proliferation and Regeneration of Glial Progenitor Cells After Injury

Summary: A new study reports Sirt1 can help glial cells to regenerate from progenitor cells in preterm babies with hypoxia related injuries. Source: Children’s National Health System. Preemie brains hold specialized cells in reserve that can repair brain injury suffered early in life. Developing brains in newborns have a sizable pool of a certain typeContinue reading “Sirt1 Regulates Proliferation and Regeneration of Glial Progenitor Cells After Injury”