Fasting starves cancer cells

Intermittent Fasting for Cancer Patients

Intermittent fasting is a style of eating with a few different variations that are all based on cycling through periods of fasting and eating normally. This kind of eating is most often used to boost weight loss, for which it has been proven effective. However, there are a number of additional health benefits from brain health to heart health, to protection against diabetes.

For patients with mesothelioma or another type of cancer, the benefits of intermittent fasting are hopeful. There is evidence from research that fasting in any form could slow tumor growth, boost the immune system, reduce treatment side effects, increase survival rates, and prevent recurrences. Research is ongoing, but for now, as long as done with medical guidance, intermittent fasting is safe and beneficial for cancer patients.

What is Intermittent Fasting?

Intermittent fasting refers to any one of a few different strategies of cycles of alternating eating and refraining from eating, or fasting. It isn’t really a diet, although some people use it to lose weight, because it does not indicate what kinds of foods you should eat. It is really more a pattern or style of eating that according to research has some real health benefits, including potentially helping cancer patients. Some of the different types of intermittent fasting include:

  • 24-Hour Fasting. This type of fasting means not eating at all for 24 hours. So, for example someone practicing this may choose to not eat between dinner one day and dinner the next day. This is typically done once or twice a week.
  • The 5:2 Diet. The 5:2 strategy modifies 24-hour fasting. It involves restricting calories for two 24-hour periods per week. On those two days women eat 500 calories and men 600.
  • The 16/8 Fast. Most popular for people using intermittent fasting to lose weight, this strategy involves not eating for 16 hours every day. Most people do this by skipping breakfast, for example, and not eating between 8:00 at night and noon the next day.
  • Calorie Restriction. This type of diet isn’t exactly fasting because there are no designated periods of not eating. But it is similar because it reduces overall calories. Calorie restriction involves reducing daily calorie intake by 20 to 40 percent every day for an extended period of time. A general guideline is 1,200 calories per day for women and 1,400 for men.

Health Benefits of Intermittent Fasting

There are significant changes that occur in the body during fasting: human growth hormone levels increase, insulin levels drop, cell repair processes speed up, and there are changes to gene expression. These changes, and potentially others, are being used to explain some of the surprising health benefits that are seen with all types of intermittent fasting and calorie restriction.

The benefit that most people turn to intermittent fasting for is weight loss. It is proven to promote weight loss and especially fat loss. This is due to lowered calorie intake, but goes beyond that with hormone changes that promote fat loss. Intermittent fasting also increases metabolic rate, making the body burn more calories.

Research is proving that there are many benefits to this style of eating that go well beyond weight loss. It lowers blood sugar levels and helps to reduce resistance to insulin, both of which protect against diabetes. Fasting is also proven to improve cardiovascular health and to promote nerve cell growth in the brain, possibly protecting against degenerative brain diseases like Alzheimer’s. In laboratory animals, intermittent fasting has been shown to slow the aging process and extend life. For cancer patients, there may be even more exciting and hopeful benefits of practicing intermittent fasting.

Triggering the Immune System for Cancer Patients

One way in which a fasting diet may help cancer patients is by triggering the immune system. The immune system is designed to target and destroy pathogens in the body, like viruses. However, it seems to be less able to find, target, and kill the body’s own abnormal cells, like cancer cells. A lot of new cancer treatments are being developed to stimulate the immune system to do this, but new research is finding that a simple fasting diet could also do it.

Once recent study from the University of Southern California was conducted using lab mice and found that when the mice received chemotherapy and a fasted diet, the immune system was better able to target and kill breast cancer cells and skin cancer cells. The mice produced more immune system cells when on the fasted diet, including the B cells and T cells that actively target and kill tumor cells. Another discovery was that cells that normally protect tumors—called T regulatory cells—were kept out of the tumors.  This may have helped chemotherapy drugs work better.

The same researchers also conducted a pilot study with human cancer patients, mainly to determine if fasting diets with chemotherapy would be safe. Both a water-only, two-day fast and a four-day, restricted-calorie diet that mimics fasting were found to be safe for cancer patients under the supervision of doctors. All of these findings indicate that fasting or a fasting-mimicked diet along with chemotherapy could be used to slow tumor growth in cancer patients.

Reducing Cancer Recurrence and Mortality Rates

Other studies have used intermittent fasting with cancer survivors to determine the effects. In one study, survivors of breast cancer practiced intermittent fasting by going for 13 hours per day without eating, a modification of the 16/8 fast. The results were reductions in cancer recurrence in the fasting group, by 36 percent. The fasting group also had better survival rates. Adding two hours to the fasting time gave even better results.

Fasting May Reduce Cancer Treatment Side Effects

Cancer treatments, especially chemotherapy, can cause side effects in patients that range from uncomfortable to debilitating. Studies have found that intermittent fasting can protect against these side effects. In one study, cancer patients fasted for a few days and then ate a normal diet before treatment. They did not lose a dangerous amount of weight or see any interference with the cancer treatments.

They did, however, see benefits in reduced side effects as compared to patients who did not fast. Patients who participated in the fasting diet experienced less fatigue and weakness, fewer headaches, less nausea, and no vomiting. They also saw reductions compared to the control group in dry mouth, mouth sores, cramps, and numbness.

Risks of Intermittent Fasting

Some critics of this style of eating worry that it promotes unhealthy eating patterns and even eating disorders. There may be a slight risk that someone will binge eat between fasting periods, for instance, which is not recommended and may reverse the beneficial effects of the diet. There are also concerns that some people may use fasting for health benefits but end up losing unhealthy amounts of weight, leading to malnourishment.

The research, however, does not support the concerns. Multiple studies have determined that intermittent fasting, when guided by doctors or other medical professionals, is safe. It is important to note that fasting and long-term calorie restriction can potentially be unsafe. Practicing any style of fasted eating should be done under the guidance of doctors, especially for people who are ill or have cancer.

Intermittent fasting may sound like a chore, but most people report that it is not difficult to do and that they feel better for it. If you are living with mesothelioma or another type of cancer, it may be worth giving a fasting diet a try. Just be sure to talk to your medical team and your oncologist before doing it and practice intermittent fasting only with medical guidance.

Page edited by Dave Foster

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Dave has been a mesothelioma Patient Advocate for over 10 years. He consistently attends all major national and international mesothelioma meetings. In doing so, he is able to stay on top of the latest treatments, clinical trials, and research results. He also personally meets with mesothelioma patients and their families and connects them with the best medical specialists and legal representatives available.Connect with Patient Advocate Dave Foster

Successful ageing

Successful ageing

The concept of successful ageing can be traced back to the 1950s and was popularised in the 1980s. Traditional definitions of successful ageing have emphasised absence of physical and cognitive disabilities.

In their 1987 article, Rowe and Kahn characterised successful ageing as involving three components:

a) freedom from disease and disability

b) high cognitive and physical functioning, and

c) social and productive engagement

Connie’s comments:

A healthy brain means healthy cognition. Which also influence our mood to be more active and learn more. Which then increases our body cells for growth and endurance, higher immune system.

When our immune system is stronger, we are less prone to disease and disability. So we ensure that we do not have anxiety but a healthy brain, helping our immune system to be health too and in return creates an environment for our body cells to grow healthy.

Aging is slowed.

Connie Dello Buono

Others wanted to be more stronger with exercise, whole foods and supplementation.

Below is an anti-aging supplement , use Distributorship/sponsor ID #: USW9578356 to order as consumer or distributor:

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LIFEPACK 22

raw carrots


 

Accumulation of changes over time

Ageing or aging (see spelling differences), is the process of becoming older. The term refers especially to human beings, many animals, and fungi, whereas for example bacteria, perennial plants and some simple animals are potentially immortal. In the broader sense, ageing can refer to single cells within an organism which have ceased dividing (cellular senescence) or to the population of a species (population ageing).
In humans, ageing represents the accumulation of changes in a human being over time,[1] encompassing physical, psychological, and social changes. Reaction time, for example, may slow with age, while knowledge of world events and wisdom may expand. Ageing is among the greatest known risk factors for most human diseases:[2] of the roughly 150,000 people who die each day across the globe, about two thirds die from age-related causes.

Connie’s comments:

Fasting, adding lemon/pinch of turmeric, ginger, baking soda and apple cider vinegar in your water can help in cleansing.

 

The causes of ageing are uncertain; current theories are assigned to the damage concept, whereby the accumulation of damage (such as DNA oxidation) may cause biological systems to fail, or to the programmed ageing concept, whereby internal processes (such as DNA methylation) may cause ageing. Programmed ageing should not be confused with programmed cell death (apoptosis).

Calorie restriction can extend lifespan by 50% in rats

The discovery, in 1934, that calorie restriction can extend lifespan by 50% in rats has motivated research into delaying and preventing ageing.

Immortal Hydra, a relative of the jellyfish

Human beings and members of other species, especially animals, necessarily experience ageing and mortality. Fungi, too, can age.[3] In contrast, many species can be considered immortal: for example, bacteria fission to produce daughter cells, strawberry plants grow runners to produce clones of themselves, and animals in the genus Hydra have a regenerative ability by which they avoid dying of old age.

Early life forms on Earth, starting at least 3.7 billion years ago,[4] were single-celled organisms. Such organisms (prokaryotes, protozoans, algae) multiply by fissioning into daughter cells; thus do not age and are innately immortal.

Every children born by a mother is minus 5 years from her life

Ageing and mortality of the individual organism became possible with the evolution of sexual reproduction,[7] which occurred with the emergence of the fungal/animal kingdoms approximately a billion years ago, and the evolution of flowering plants 160 million years ago. The sexual organism could henceforth pass on some of its genetic material to produce new individuals and could itself become disposable with respect to the survival of its species.

This classic biological idea has however been perturbed recently by the discovery that the bacterium E. coli may split into distinguishable daughter cells, which opens the theoretical possibility of “age classes” among bacteria.

Stem cells

Even within humans and other mortal species, there are cells with the potential for immortality: cancer cells which have lost the ability to die when maintained in a cell culture such as the HeLa cell line,[9] and specific stem cells such as germ cells (producing ova and spermatozoa).[10] In artificial cloning, adult cells can be rejuvenated to embryonic status and then used to grow a new tissue or animal without ageing.[11] Normal human cells however die after about 50 cell divisions in laboratory culture (the Hayflick Limit, discovered by Leonard Hayflick in 1961).

Effects of ageing

Enlarged ears and noses of old humans are sometimes blamed on continual cartilage growth, but the cause is more probably gravity.[12]

Comparison of a normal aged brain (left) and a brain affected by Alzheimer’s disease (right).

A number of characteristic ageing symptoms are experienced by a majority or by a significant proportion of humans during their lifetimes.

  • Teenagers lose the young child’s ability to hear high-frequency sounds above 20 kHz.
  • Cognitive decline begins in the mid-20s.
  • Wrinkles develop mainly due to photoageing, particularly affecting sun-exposed areas (face). Sun Damage, lack of sleep, alcohol, over medication and other factors contribute to aging skin

After peaking in the mid-20s, female fertility declines.

People over 35 years of age are at risk for developing presbyopia.[18][19] and most people benefit from reading glasses by age 45–50.[20] The cause is lens hardening by decreasing levels of α-crystallin, a process which may be sped up by higher temperatures.
Around age 50, hair turns grey.

Pattern hair loss by the age of 50 affects about half of males and a quarter of females.

Menopause typically occurs between 49 and 52 years of age.

In the 60–64 age cohort, the incidence of osteoarthritis rises to 53%. Only 20% however report disabling osteoarthritis at this age.
Almost half of people older than 75 have hearing loss inhibiting spoken communication.

Cataract at age 80

By age 80, more than half of all Americans either have a cataract or have had cataract surgery.

Loss of muscle mass can be slowed by exercise

Frailty, defined as loss of muscle mass and mobility, affects 25% of those over 85.[28][29]
Atherosclerosis is classified as an ageing disease.[30] It leads to cardiovascular disease (for example stroke and heart attack)[31] which globally is the most common cause of death.

Can we live until 115 years old?

The maximum human lifespan is suggested to be 115 years “for the foreseeable future”.[33][34] The oldest reliably recorded human was Jeanne Calment who attained 122 years and died in 1997.

Dementia

Dementia becomes more common with age. About 3% of people between the ages of 65 and 74, 19% between 75 and 84, and nearly half of those over 85 years of age have dementia.[36] The spectrum ranges from mild cognitive impairment to the neurodegenerative diseases of Alzheimer’s disease, cerebrovascular disease, Parkinson’s disease and Lou Gehrig’s disease. Furthermore, many types of memory may decline with ageing, but not semantic memory or general knowledge such as vocabulary definitions, which typically increases or remains steady until late adulthood[37] (see Ageing brain). Intelligence may decline with age, though the rate may vary depending on the type and may in fact remain steady throughout most of the lifespan, dropping suddenly only as people near the end of their lives. Individual variations in rate of cognitive decline may therefore be explained in terms of people having different lengths of life.[38] There might be changes to the brain: after 20 years of age there may be a 10% reduction each decade in the total length of the brain’s myelinated axons.

Age can result in visual impairment, whereby non-verbal communication is reduced,which can lead to isolation and possible depression. Macular degeneration causes vision loss and increases with age, affecting nearly 12% of those above the age of 80.[42] This degeneration is caused by systemic changes in the circulation of waste products and by growth of abnormal vessels around the retina.
A distinction can be made between “proximal ageing” (age-based effects that come about because of factors in the recent past) and “distal ageing” (age-based differences that can be traced to a cause in person’s early life, such as childhood poliomyelitis).[38]
Ageing is among the greatest known risk factors for most human diseases.[2] Of the roughly 150,000 people who die each day across the globe, about two thirds—100,000 per day—die from age-related causes. In industrialised nations, the proportion is higher, reaching 90%.

95-year-old woman holding a five-month-old boy

At present, researchers are only just beginning to understand the biological basis of ageing even in relatively simple and short-lived organisms such as yeast.[47] Less still is known of mammalian ageing, in part due to the much longer lives of even small mammals such as the mouse (around 3 years). A model organism for studying of ageing is the nematode C. elegans, thanks to its short lifespan of 2–3 weeks, our ability to easily perform genetic manipulations or to suppress gene activity with RNA interference, or other factors.[48] Most known mutations and RNA interference targets that extend lifespan were first discovered in C. elegans.
The factors proposed to influence biological ageing[50] fall into two main categories, programmed and damage-related. Programmed factors follow a biological timetable, perhaps one that might be a continuation of the one that regulates childhood growth and development. This regulation would depend on changes in gene expression that affect the systems responsible for maintenance, repair and defence responses. Damage-related factors include internal and environmental assaults to living organisms that induce cumulative damage at various levels.
There are three main metabolic pathways which can influence the rate of ageing, discussed below:

  1. the FOXO3/Sirtuin pathway, probably responsive to caloric restriction
  2. the Growth hormone/Insulin-like growth factor 1 signalling pathway
  3. the activity levels of the electron transport chain in mitochondria[52] and (in plants) in chloroplasts.

It is likely that most of these pathways affect ageing separately, because targeting them simultaneously leads to additive increases in lifespan.

Programmed factors

The rate of ageing varies substantially across different species, and this, to a large extent, is genetically based. For example, numerous perennial plants ranging from strawberries and potatoes to willow trees typically produce clones of themselves by vegetative reproduction and are thus potentially immortal, while annual plants such as wheat and watermelons die each year and reproduce by sexual reproduction. In 2008 it was discovered that inactivation of only two genes in the annual plant Arabidopsis thaliana leads to its conversion into a potentially immortal perennial plant.

Senescence

Clonal immortality apart, there are certain species whose individual lifespans stand out among Earth’s life-forms, including the bristlecone pine at 5062 years[55] (however Hayflick states that the bristlecone pine has no cells older than 30 years), invertebrates like the hard clam (known as quahog in New England) at 508 years,[56] the Greenland shark at 400 years,[57] fish like the sturgeon and the rockfish, and the sea anemone[58] and lobster.[59][60] Such organisms are sometimes said to exhibit negligible senescence.[61] The genetic aspect has also been demonstrated in studies of human centenarians.
In laboratory settings, researchers have demonstrated that selected alterations in specific genes can extend lifespan quite substantially in yeast and roundworms, less so in fruit flies and less again in mice. Some of the targeted genes have homologues across species and in some cases have been associated with human longevity.

DNA methylation

The strong effect of age on DNA methylation levels has been known since the late 1960s.[63] Horvath hypothesised that DNA methylation age measures the cumulative effect of an epigenetic maintenance system but details are unknown. DNA methylation age of blood predicts all-cause mortality in later life.[64][65][66] Furthermore, prematurely aged mice can be rejuvenated and their lives extended by 30% by partially “resetting” the methylation pattern in their cells (a full reset leads to undesirable immortal cancer cells). This resetting into a juvenile state was experimentally achieved by activating the four Yamanaka DNA transcription factors – Sox2, Oct4, Klf4 and c-Myc (which have previously been routinely used for producing young animals from cloned adult skin cells).[67][68]
Telomeres: In humans and other animals, cellular senescence has been attributed to the shortening of telomeres at each cell division;[69] when telomeres become too short, the cells senesce and die or cease multiplying.[70] The length of telomeres is therefore the “molecular clock”, predicted by Hayflick.[71][72] However, telomere length in wild mouse strains is unrelated to lifespan,[73] and mice lacking the telomerase enzyme do not have a dramatically reduced lifespan.[74] Laboratory mice’s telomeres are many times longer than human ones.[75] Another caveat is that a study following nearly 1000 humans for ten years showed that while some humans do shorten their telomeres over time, a third of the participants did not.
A variation in the gene FOXO3A has a positive effect on the life expectancy of humans, and is found much more often in people living to 100 and beyond – moreover, this appears to be true worldwide.[77][78] FOXO3A acts on the sirtuin family of genes which also have a significant effect on lifespan in yeast and in nematodes. Sirtuin in turn inhibits mTOR.

Caloric restriction

Caloric restriction leads to longer lifespans in various species, an effect that is unclear,[53] but probably mediated by the nutrient-sensing function of the mTOR pathway.[80]
mTOR, a protein that inhibits autophagy, has been linked to ageing through the insulin signalling pathway. mTOR functions through nutrient and growth cues leading scientists to believe that dietary restriction and mTOR are related in terms of longevity. When organisms restrict their diet, mTOR activity is reduced, which allows an increased level of autophagy. This recycles old or damaged cell parts, which increases longevity and decreases the chances of being obese. This is thought to prevent spikes of glucose concentration in the blood, leading to reduced insulin signalling. This has been linked to less mTOR activation as well. Therefore, longevity has been connected to caloric restriction and insulin sensitivity inhibiting mTOR, which in turns allows autophagy to occur more frequently. It may be that mTOR inhibition and autophagy reduce the effects of reactive oxygen species on the body, which damage DNA and other organic material, so longevity would be increased.

A decreased Growth hormone/Insulin-like Growth Factor 1 signalling pathway has been associated with increased life span in various organisms including fruit flies, nematodes and mice. The precise mechanism by which decreased GH/IGF-1 signalling increases longevity is unknown, but various mouse strains with decreased GH and/or IGF-1 induced signalling share a similar phenotype which includes increased insulin sensitivity, enhanced stress resistance and protection from carcinogenesis. The studied mouse strains with decreased GH signalling showed between 20% and 68% increased longevity, and mouse strains with decreased IGF-1 induced signalling revealed a 19 to 33% increase in life span when compared to control mice.[82]
Over-expression of the Ras2 gene increases lifespan in yeast by 30%.[83] A yeast mutant lacking the genes sch9 and ras2 has recently been shown to have a tenfold increase in lifespan under conditions of calorie restriction and is the largest increase achieved in any organism.
Evolutionary theories of ageing: Many have argued that life span, like other phenotypes, is selected. Traits that benefit early survival and reproduction will be selected for even if they contribute to an earlier death. Such a genetic effect is called the antagonistic pleiotropy effect when referring to a gene (pleiotropy signifying the gene has a double function – enabling reproduction at a young age but costing the organism life expectancy in old age) and is called the disposable soma effect when referring to an entire genetic programme (the organism diverting limited resources from maintenance to reproduction).[7] The biological mechanisms which regulate lifespan evolved several hundred million years ago.

Oxygen-deprived bacterial culture

Some evidence is provided by oxygen-deprived bacterial cultures.
The theory would explain why the autosomal dominant disease, Huntington’s disease, can persist even though it is inexorably lethal. Also, it has been suggested that some of the genetic variants that increase fertility in the young increase cancer risk in the old. Such variants occur in genes p53[87] and BRCA1.

Reproductive hormones

The reproductive-cell cycle theory argues that ageing is regulated specifically by reproductive hormones that act in an antagonistic pleiotropic manner via cell cycle signalling, promoting growth and development early in life to achieve reproduction, but becoming dysregulated later in life, driving senescence (dyosis) in a futile attempt to maintain reproductive ability.[1][89] The endocrine dyscrasia that follows the loss of follicles with menopause, and the loss of Leydig and Sertoli cells during andropause, drive aberrant cell cycle signalling that leads to cell death and dysfunction, tissue dysfunction (disease) and ultimately death.

Moreover, the hormones that regulate reproduction also regulate cellular metabolism, explaining the increases in fat deposition during pregnancy through to the deposition of centralised adiposity with the dysregulation of the HPG axis following menopause and during andropause (Atwood and Bowen, 2006). This theory, which introduced a new definition of ageing, has facilitated the conceptualisation of why and how ageing occurs at the evolutionary, physiological and molecular levels.

Autoimmunity

The idea that ageing results from an increase in autoantibodies that attack the body’s tissues. A number of diseases associated with ageing, such as atrophic gastritis and Hashimoto’s thyroiditis, are probably autoimmune in this way. However, while inflammation is very much evident in old mammals, even completely immunodeficient mice raised in pathogen-free laboratory conditions still experience senescence.[citation needed]

An elderly Somali woman

The cellular balance between energy generation and consumption (energy homeostasis) requires tight regulation during ageing. In 2011, it was demonstrated that acetylation levels of AMP-activated protein kinase change with age in yeast and that preventing this change slows yeast ageing.

Damage-related factors

DNA damage theory of ageing: DNA damage is thought to be the common basis of both cancer and ageing, and it has been argued that intrinsic causes of DNA damage are the most important drivers of ageing.[91][92][93] Genetic damage (aberrant structural alterations of the DNA), mutations (changes in the DNA sequence), and epimutations (methylation of gene promoter regions or alterations of the DNA scaffolding which regulate gene expression), can cause abnormal gene expression. DNA damage causes the cells to stop dividing or induces apoptosis, often affecting stem cell pools and hence hindering regeneration. However, lifelong studies of mice suggest that most mutations happen during embryonic and childhood development, when cells divide often, as each cell division is a chance for errors in DNA replication.

Genetic instability

In heart muscle cells, dogs annually lose approximately 3.3% of the DNA in their heart muscle cells while humans lose approximately 0.6% of their heart muscle DNA each year. These numbers are close to the ratio of the maximum longevities of the two species (120 years vs. 20 years, a 6/1 ratio). The comparative percentage is also similar between the dog and human for yearly DNA loss in the brain and lymphocytes. As stated by lead author, Bernard L. Strehler, “… genetic damage (particularly gene loss) is almost certainly (or probably the) central cause of ageing.”[95]

Accumulation of waste

A buildup of waste products in cells presumably interferes with metabolism. For example, a waste product called lipofuscin is formed by a complex reaction in cells that binds fat to proteins. This waste accumulates in the cells as small granules, which increase in size as a person ages.
The hallmark of ageing yeast cells appears to be overproduction of certain proteins.[47]
Autophagy induction can enhance clearance of toxic intracellular waste associated with neurodegenerative diseases and has been comprehensively demonstrated to improve lifespan in yeast, worms, flies, rodents and primates. The situation, however, has been complicated by the identification that autophagy up-regulation can also occur during ageing.[97] Autophagy is enhanced in obese mice by caloric restriction, exercise, and a low fat diet (but in these mice is evidently not related with the activation of AMP-activated protein kinase, see above).

Wear-and-tear theory

The very general idea that changes associated with ageing are the result of chance damage that accumulates over time.[99]
Accumulation of errors: The idea that ageing results from chance events that escape proof reading mechanisms, which gradually damages the genetic code.
Cross-linkage: The idea that ageing results from accumulation of cross-linked compounds that interfere with normal cell function.[72][100]
Studies of mtDNA mutator mice have shown that increased levels of somatic mtDNA mutations directly can cause a variety of ageing phenotypes. The authors propose that mtDNA mutations lead to respiratory-chain-deficient cells and thence to apoptosis and cell loss.

They cast doubt experimentally however on the common assumption that mitochondrial mutations and dysfunction lead to increased generation of reactive oxygen species (ROS).

Free-radical theory

Damage by free radicals, or more generally reactive oxygen species or oxidative stress, create damage that may gives rise to the symptoms we recognise as ageing.[72][102] Michael Ristow’s group has provided evidence that the effect of calorie restriction may be due to increased formation of free radicals within the mitochondria, causing a secondary induction of increased antioxidant defence capacity.[103]
DNA oxidation and caloric restriction: Caloric restriction reduces 8-OH-dG DNA damage in organs of ageing rats and mice.[104][105] Thus, reduction of oxidative DNA damage is associated with a slower rate of ageing and increased lifespan[106].

Lifestyle

Caloric restriction substantially affects lifespan in many animals, including the ability to delay or prevent many age-related diseases.[107] Typically, this involves caloric intake of 60–70% of what an ad libitum animal would consume, while still maintaining proper nutrient intake.[107] In rodents, this has been shown to increase lifespan by up to 50%;[108] similar effects occur for yeast and Drosophila.[107] No lifespan data exist for humans on a calorie-restricted diet,[82] but several reports support protection from age-related diseases.[109][110] Two major ongoing studies on rhesus monkeys initially revealed disparate results; while one study, by the University of Wisconsin, showed that caloric restriction does extend lifespan,[111] the second study, by the National Institute on Ageing (NIA), found no effects of caloric restriction on longevity.[112] Both studies nevertheless showed improvement in a number of health parameters. Notwithstanding the similarly low calorie intake, the diet composition differed between the two studies (notably a high sucrose content in the Wisconsin study), and the monkeys have different origins (India, China), initially suggesting that genetics and dietary composition, not merely a decrease in calories, are factors in longevity.[82] However, in a comparative analysis in 2014, the Wisconsin researchers found that the allegedly non-starved NIA control monkeys in fact are moderately underweight when compared with other monkey populations, and argued this was due to the NIA’s apportioned feeding protocol in contrast to Wisconsin’s truly unrestricted ad libitum feeding protocol.[113] They conclude that moderate calorie restriction rather than extreme calorie restriction is sufficient to produce the observed health and longevity benefits in the studied rhesus monkeys.
In his book How and Why We Age, Hayflick says that caloric restriction may not be effective in humans, citing data from the Baltimore Longitudinal Study of Aging which shows that being thin does not favour longevity.[need quotation to verify][115] Similarly, it is sometimes claimed that moderate obesity in later life may improve survival, but newer research has identified confounding factors such as weight loss due to terminal disease. Once these factors are accounted for, the optimal body weight above age 65 corresponds to a leaner body mass index of 23 to 27.[116]
Alternatively, the benefits of dietary restriction can also be found by changing the macro nutrient profile to reduce protein intake without any changes to calorie level, resulting in similar increases in longevity.[117][118] Dietary protein restriction not only inhibits mTOR activity but also IGF-1, two mechanisms implicated in ageing.[80] Specifically, reducing leucine intake is sufficient to inhibit mTOR activity, achievable through reducing animal food consumption.[119][120]
The Mediterranean diet is credited with lowering the risk of heart disease and early death.[121][122] The major contributors to mortality risk reduction appear to be a higher consumption of vegetables, fish, fruits, nuts and monounsaturated fatty acids, i.e., olive oil.[123]

Sleep

The amount of sleep has an impact on mortality. People who live the longest report sleeping for six to seven hours each night.[124][125] Lack of sleep (<5 hours) more than doubles the risk of death from cardiovascular disease, but too much sleep (>9 hours) is associated with a doubling of the risk of death, though not primarily from cardiovascular disease.[126] Sleeping more than 7 to 8 hours per day has been consistently associated with increased mortality, though the cause is probably other factors such as depression and socioeconomic status, which would correlate statistically.[127] Sleep monitoring of hunter-gatherer tribes from Africa and from South America has shown similar sleep patterns across continents: their average sleeping duration is 6.4 hours (with a summer/winter difference of 1 hour), afternoon naps (siestas) are uncommon, and insomnia is very rare (tenfold less than in industrial societies).[128]

Physical exercise

Physical exercise may increase life expectancy.[129] People who participate in moderate to high levels of physical exercise have a lower mortality rate compared to individuals who are not physically active.[130] Moderate levels of exercise have been correlated with preventing aging and improving quality of life by reducing inflammatory potential.[131] The majority of the benefits from exercise are achieved with around 3500 metabolic equivalent (MET) minutes per week.[132] For example, climbing stairs 10 minutes, vacuuming 15 minutes, gardening 20 minutes, running 20 minutes, and walking or bicycling for 25 minutes on a daily basis would together achieve about 3000 MET minutes a week.

Chronic stress

Avoidance of chronic stress (as opposed to acute stress) is associated with a slower loss of telomeres in most but not all studies,[133][134] and with decreased cortisol levels. A chronically high cortisol level compromises the immune system, causes cardiac damage/arterosclerosis and is associated with facial ageing, and the latter in turn is a marker for increased morbidity and mortality.[135][136] Stress can be countered by social connection, spirituality, and (for men more clearly than for women) married life, all of which are associated with longevity.[137][138][139]
Medical intervention
The following drugs and interventions have been shown to retard or reverse the biological effects of ageing in animal models, but none has yet been proven to do so in humans.

Resveratrol

Evidence in both animals and humans suggests that resveratrol may be a caloric restriction mimetic.[140]
As of 2015 metformin was under study for its potential effect on slowing ageing in the worm C.elegans and the cricket.[141] Its effect on otherwise healthy humans is unknown.[141]
Rapamycin was first shown to extend lifespan in eukaryotes in 2006 by Powers et al. who showed a dose-responsive effect of rapamycin on lifespan extension in yeast cells.[142] In a 2009 study, the lifespans of mice fed rapamycin were increased between 28 and 38% from the beginning of treatment, or 9 to 14% in total increased maximum lifespan. Of particular note, the treatment began in mice aged 20 months, the equivalent of 60 human years.[143] Rapamycin has subsequently been shown to extend mouse lifespan in several separate experiments,[144][145] and is now being tested for this purpose in nonhuman primates (the marmoset monkey).[146]
Cancer geneticist Ronald A. DePinho and his colleagues published research in mice where telomerase activity was first genetically removed. Then, after the mice had prematurely aged, they restored telomerase activity by reactivating the telomerase gene. As a result, the mice were rejuvenated: Shrivelled testes grew back to normal and the animals regained their fertility. Other organs, such as the spleen, liver, intestines and brain, recuperated from their degenerated state. “[The finding] offers the possibility that normal human ageing could be slowed by reawakening the enzyme in cells where it has stopped working” says Ronald DePinho. However, activating telomerase in humans could potentially encourage the growth of tumours.[147]
Most known genetic interventions in C. elegans increase lifespan by 1.5 to 2.5-fold. As of 2009, the record for lifespan extension in C. elegans is a single-gene mutation which increases adult survival by tenfold.[49] The strong conservation of some of the mechanisms of ageing discovered in model organisms imply that they may be useful in the enhancement of human survival. However, the benefits may not be proportional; longevity gains are typically greater in C. elegans than fruit flies, and greater in fruit flies than in mammals. One explanation for this is that mammals, being much longer-lived, already have many traits which promote lifespan.[49]

Different cultures

Different cultures express age in different ways. The age of an adult human is commonly measured in whole years since the day of birth. Arbitrary divisions set to mark periods of life may include: juvenile (via infancy, childhood, preadolescence, adolescence), early adulthood, middle adulthood, and late adulthood. More casual terms may include “teenagers,” “tweens,” “twentysomething”, “thirtysomething”, etc. as well as “denarian”, “vicenarian”, “tricenarian”, “quadragenarian”, etc.

Most legal systems define a specific age for when an individual is allowed or obliged to do particular activities. These age specifications include voting age, drinking age, age of consent, age of majority, age of criminal responsibility, marriageable age, age of candidacy, and mandatory retirement age. Admission to a movie for instance, may depend on age according to a motion picture rating system. A bus fare might be discounted for the young or old. Each nation, government and non-governmental organisation has different ways of classifying age. In other words, chronological ageing may be distinguished from “social ageing” (cultural age-expectations of how people should act as they grow older) and “biological ageing” (an organism’s physical state as it ages).[159]
In a UNFPA report about ageing in the 21st century, it highlighted the need to “Develop a new rights-based culture of ageing and a change of mindset and societal attitudes towards ageing and older persons, from welfare recipients to active, contributing members of society.”[160] UNFPA said that this “requires, among others, working towards the development of international human rights instruments and their translation into national laws and regulations and affirmative measures that challenge age discrimination and recognise older people as autonomous subjects.”[160] Older persons make contributions to society including caregiving and volunteering. For example, “A study of Bolivian migrants who [had] moved to Spain found that 69% left their children at home, usually with grandparents. In rural China, grandparents care for 38% of children aged under five whose parents have gone to work in cities.”[160]

Population ageing

A map showing median age figures for 2015
Population ageing is the increase in the number and proportion of older people in society. Population ageing has three possible causes: migration, longer life expectancy (decreased death rate) and decreased birth rate. Ageing has a significant impact on society. Young people tend to have fewer legal privileges (if they are below the age of majority), they are more likely to push for political and social change, to develop and adopt new technologies, and to need education. Older people have different requirements from society and government, and frequently have differing values as well, such as for property and pension rights.
In the 21st century, one of the most significant population trends is ageing.[162] Currently, over 11% of the world’s current population are people aged 60 and older and the United Nations Population Fund (UNFPA) estimates that by 2050 that number will rise to approximately 22%.[160] Ageing has occurred due to development which has enabled better nutrition, sanitation, health care, education and economic well-being. Consequently, fertility rates have continued to decline and life expectancy have risen. Life expectancy at birth is over 80 now in 33 countries. Ageing is a “global phenomenon,” that is occurring fastest in developing countries, including those with large youth populations, and poses social and economic challenges to the work which can be overcome with “the right set of policies to equip individuals, families and societies to address these challenges and to reap its benefits.”
As life expectancy rises and birth rates decline in developed countries, the median age rises accordingly. According to the United Nations, this process is taking place in nearly every country in the world.[164] A rising median age can have significant social and economic implications, as the workforce gets progressively older and the number of old workers and retirees grows relative to the number of young workers. Older people generally incur more health-related costs than do younger people in the workplace and can also cost more in worker’s compensation and pension liabilities.[165] In most developed countries an older workforce is somewhat inevitable. In the United States for instance, the Bureau of Labor Statistics estimates that one in four American workers will be 55 or older by 2020.
Among the most urgent concerns of older persons worldwide is income security. This poses challenges for governments with ageing populations to ensure investments in pension systems continues in order to provide economic independence and reduce poverty in old age. These challenges vary for developing and developed countries. UNFPA stated that, “Sustainability of these systems is of particular concern, particularly in developed countries, while social protection and old-age pension coverage remain a challenge for developing countries, where a large proportion of the labour force is found in the informal sector.”
The global economic crisis has increased financial pressure to ensure economic security and access to health care in old age. In order to elevate this pressure “social protection floors must be implemented in order to guarantee income security and access to essential health and social services for all older persons and provide a safety net that contributes to the postponement of disability and prevention of impoverishment in old age.”[160]
It has been argued that population ageing has undermined economic development.[166] Evidence suggests that pensions, while making a difference to the well-being of older persons, also benefit entire families especially in times of crisis when there may be a shortage or loss of employment within households. A study by the Australian Government in 2003 estimated that “women between the ages of 65 and 74 years contribute A$16 billion per year in unpaid caregiving and voluntary work. Similarly, men in the same age group contributed A$10 billion per year.”[160]
Due to increasing share of the elderly in the population, health care expenditures will continue to grow relative to the economy in coming decades. This has been considered as a negative phenomenon and effective strategies like labour productivity enhancement should be considered to deal with negative consequences of ageing.[167]

Sociology

In the field of sociology and mental health, ageing is seen in five different views: ageing as maturity, ageing as decline, ageing as a life-cycle event, ageing as generation, and ageing as survival.[168] Positive correlates with ageing often include economics, employment, marriage, children, education, and sense of control, as well as many others. The social science of ageing includes disengagement theory, activity theory, selectivity theory, and continuity theory. Retirement, a common transition faced by the elderly, may have both positive and negative consequences.[169] As cyborgs currently are on the rise some theorists argue there is a need to develop new definitions of ageing and for instance a bio-techno-social definition of ageing has been suggested.[170]
Health care demand
With age inevitable biological changes occur that increase the risk of illness and disability. UNFPA states that,[163]
“A life-cycle approach to health care – one that starts early, continues through the reproductive years and lasts into old age – is essential for the physical and emotional well-being of older persons, and, indeed, all people. Public policies and programmes should additionally address the needs of older impoverished people who cannot afford health care.”
Many societies in Western Europe and Japan have ageing populations. While the effects on society are complex, there is a concern about the impact on health care demand. The large number of suggestions in the literature for specific interventions to cope with the expected increase in demand for long-term care in ageing societies can be organised under four headings: improve system performance; redesign service delivery; support informal caregivers; and shift demographic parameters.[171]
However, the annual growth in national health spending is not mainly due to increasing demand from ageing populations, but rather has been driven by rising incomes, costly new medical technology, a shortage of health care workers and informational asymmetries between providers and patients.[172] A number of health problems become more prevalent as people get older. These include mental health problems as well as physical health problems, especially dementia.
It has been estimated that population ageing only explains 0.2 percentage points of the annual growth rate in medical spending of 4.3% since 1970. In addition, certain reforms to the Medicare system in the United States decreased elderly spending on home health care by 12.5% per year between 1996 and 2000.[173]
Self-perception of ageing
Positive self-perception of health has been correlated with higher well-being and reduced mortality in the elderly.[174][175] Various reasons have been proposed for this association; people who are objectively healthy may naturally rate their health better than that of their ill counterparts, though this link has been observed even in studies which have controlled for socioeconomic status, psychological functioning and health status.[176] This finding is generally stronger for men than women,[175] though this relationship is not universal across all studies and may only be true in some circumstances.[176]
As people age, subjective health remains relatively stable, even though objective health worsens.[177] In fact, perceived health improves with age when objective health is controlled in the equation.[178] This phenomenon is known as the “paradox of ageing.” This may be a result of social comparison;[179] for instance, the older people get, the more they may consider themselves in better health than their same-aged peers.[180] Elderly people often associate their functional and physical decline with the normal ageing process.[181][182]

 

Cultural references

The ancient Greek dramatist Euripides (5th century BC) describes the multiply-headed mythological monster Hydra as having a regenerative capacity which makes it immortal, which is the historical background to the name of the biological genus Hydra. The Book of Job (c. 6th century BC) describes human lifespan as inherently limited and makes a comparison with the innate immortality that a felled tree may have when undergoing vegetative regeneration.[185]
See also

 


Lectin, gluten, stomach, fasting, toxins, wheat, and foods

By Dr Mercola

From an evolutionary standpoint, any creature, including plants, has a built-in imperative to grow, thrive and propagate. Plants, being rooted into the ground, cannot outrun a predatory insect. Instead, plants use chemistry for self-defense. One of the plant kingdom’s self-defense systems is lectins — not to be confused with lecithin or leptin.

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Lectins are plant proteins, sometimes called sticky proteins or glyca-binding proteins, because they seek out and bind to certain sugar molecules on the surface of cells. There are many types of lectins, and the main difference between them is the type of sugar each prefers and binds to.

Some — including wheat germ agglutinin (WGA), found in wheat and other grass-family seeds — bind to specific receptor sites on your intestinal mucosal cells and interfere with the absorption of nutrients across your intestinal wall. As such, they act as “antinutrients,” and can have a detrimental effect on your gut microbiome by shifting the balance of your bacterial flora — a common precursor to leaky gut.

“I like to think of it as they hack into our communication system, or any predator’s communication system,” Gundry says. “For instance, in insects, they attack a sugar called sialic acid which, among other things, sits between the endings of nerves. One nerve talks to the other nerve by acetylcholine jumping through that space.

Sialic acid allows that to happen. Lectins bind to sialic acid and so interrupt nerve transmission. If you think about it, paralyzing an insect is a great defense system because if the insect can’t move, bingo, you’ve solved the problem. One of the things I’ve learned through the years through my patients is we’re just a giant insect to a plant.

What may happen to an insect fairly instantaneously by eating some plant lectins may take years in us, who are giant insects, to manifest. It may manifest as neuropathy, it may manifest as brain fog, arthritis or heart disease. But the longer I do this, the more I’m convinced that almost every disease process … can be traced back to … plant lectins.

That’s a long-winded explanation for how plants don’t like us. They absolutely don’t want to be eaten. They’ve had 400 million years to work out defense systems — a really long time.”

The Role of Your Microbiome

One of the things that struck me about Gundry’s approach is that it targets the mitochondria and the microbiome, both of which are vital for optimal health. Few physicians, even those in the integrative medicine field, fully understand the importance of mitochondrial function, but Gundry certainly does. And, while the human genome has received a majority of the scientific attention, the bacterial microbiome genome is actually far more important. As noted by Gundry:

“Our microbiome is, I think, our early warning system, because about 99 percent of all the genes that make up [the human body] are actually nonhuman, they’re bacterial, viral and fungal … [from which] we’ve uploaded most of the information about interacting with our environment … because the microbiome is capable of almost instantaneous changing and information processing that we actually don’t have the ability to do.

We’re beginning to realize … that the microbiome is not only how we interact with plant materials … like lectins, but probably more importantly, our microbiome teaches our immune system whether a particular plant compound is a friend or foe [based on] how long we’ve known that plant compound.

There are lectins in everything. But the longer we’ve interacted with lectins and the longer our microbiome has interacted with them, the more our microbiome kind of tells our immune system, ‘Hey, guys, it’s cool. We’ve known these guys for 40 million years. Chill out. They’re a pain, but we can handle them.’

From an evolutionary perspective, if you look at modern foods — say the grains and the beans, which we started interacting with 10,000 years ago, which is a blink of time — our microbiome [regards them as] foreign substances … [T]here’s no lectin speed dating in evolution.”

The Importance of Mitochondrial Function

With regard to mitochondria, “mitochondrial flexibility is one of the unique things that make us human,” Gundry says, comparing the human race to a “fat-storing ape.” Whether you ascribe to the evolutionary theory or not, humans and apes have many genetic similarities, but the ability to store fat is a unique human feature. No other great apes can do that.

Chimps, gorillas and orangutans carry 3 percent body fat. Few humans could ever achieve that low of a body fat percentage unless we were near death from starvation.

“The reason we’re designed to [store fat is to] be able to access fat for fuel,” Gundry says. “The reason why [humans] have been able to take over all parts of the world … [is] because we can cycle back and forth, having our mitochondria use fat for fuel or glucose for fuel. We’re designed to shift very quickly … even within 24 hours.

[Most people] no longer have that metabolic flexibility [because] we’ve been constantly bombarding our mitochondria with an overload of glucose as a fuel, and that really underlies, I think, most disease processes.”

How Intermittent Fasting Boosts Mitochondrial Flexibility

One of the strategies Gundry recommends and uses to improve his own metabolic flexibility is intermittent fasting. For nearly a decade now, he’s been fasting for 22 hours a day, five days a week, from January through June 1, which means he eats all his calories for the day during a two-hour window. On the weekends, he eats lunch and dinner.

“I don’t eat breakfast. I don’t eat lunch. I eat my calories between 6 and 8 o’clock at night. I do that because my wife and I are at home at that time. If I was really smart, I would [eat] earlier in the day, but, you know, you’ve got to be practical in one way or another …

In summer, I’ll have a smoothie with some MCT oil in it, half an avocado, some romaine lettuce, spinach, half a lemon and a little bit of vanilla or stevia. Then I won’t eat lunch. At dinner, same sort of thing, I try to pack all of my calories in between 6 and 8 o’clock at night … 

[June 1], I finished my winter fast, if you will. Now, why do I do that? [Historically], food was a rare thing to find [during the winter]. Again, our metabolic advantage is we’re really good at starvation. It’s what allowed us to survive.

We know that during food scarcity, not only do our mitochondria rev up, but more importantly, our entire immune system and genetic monitoring basically says, ‘Look, times are tough. We don’t know when the next good food supply is going to come. We’ve got to make it through to that next period. We’re going to look at every cell in our body. We’re going to look at whether they’re pulling their own weight.

Are they odd? Are they not very fuel-efficient? We’re going to jettison that. We’re going to create apoptosis until these cells commit suicide.’ It’s kind of like if we were in a hot air balloon and we’re heading for the mountain and we’re going to crash, we’ve got to start throwing things overboard to get more lift.

I think that’s a fundamental principle that you’ve known for a number of years and that I’ve certainly preached for a number of years. The more we understand that that’s how successful aging occurs and study successful agers, one of the things that’s fascinating, particularly in an animal model, is that this intermittent fasting, this challenging [your mitochondria], is the way to do it.”

Although I used to do 14- to 16-hour intermittent fasts, because I felt that it was wise to increase glycogen stores prior to strength training, I have come to realize that’s not true. In fact, it’s counterproductive, as carbs after strength training can increase insulin and diminish IGF-1 response and blunt the anabolic stimulus. So now I am fasting for 18 to 20 hours a day and do all my strength training in a fasted state.

That may sound challenging, but I can confidently assure you, from personal experience, that once you are fat adapted there are no cravings. Additionally, I recently interviewed Dr. Dale Bredesen, who wrote the book “The End of Alzheimer’s: The First Program to Prevent and Reverse Cogntive Decline,” in which he discusses how ApoE4 is a genetic risk factor for Alzheimer’s but ONLY if you don’t intermittent fast. If you do, it will likely actually decrease your risk for the disease as its biological function is to allow us to go for longer periods of time without food.

The Importance of Ketogenic Cycling

Gundry also understands the importance of cycling in and out of nutritional ketosis. While your body is still burning sugar as its primary fuel, you’ll want to be quite strict about not going over your net carb allotment. But once your body has regained the metabolic flexibility to burn fat, it’s really important to cycle in and out or on and off.

I suggest doubling, tripling or even quadrupling your net carbs two days a week, because the metabolic “magic” actually happens during the refeeding phase. As noted by Gundry:

“You have to look at it evolutionarily. It really was feast or famine. When we hit large amounts of food, whether it was a fruit tree or whether it was honey or a wildebeest or a mastodon, there was no food storage system. People tend to forget that nobody walked out of their cave and said, ‘What’s for breakfast?’ There was no refrigerator to have organic berries in every day.

When we chanced upon fuel, then our beautiful design [allowed us to] eat large quantities of [food] and store it as fat. Because, very shortly, whether it was a period of drought, whether it was a period of winter, we were going to regress. I’d like people to think of circadian rhythms. Obviously, we have a 24-hour clock. We have a moon clock. We have seasonal clocks.

What I like people to think of is that we have a period of every year where [we’re in] a growth cycle … That’s the time of growth and it’s a time to reproduce. Then there’s a time of involution, whether it’s a tree dropping its leaves, whether it’s an animal hibernating.

That’s the time where we kind of take stock of everything. That yin and yang, that flow that would happen every year on seasonal basis has completely been lost. We have to have periods where we consume excess calories, then we have to have periods where the exact opposite happens.

Years ago, after my first book came out, I was invited to Phoenix, Arizona, by a blogger named John Kiefer. Kiefer said you should burn fat for fuel most of the time. But every week, you should have what’s called “carb nite loading.” He chanced upon this by accident, but he made a career out of it. I picked his brain and he picked my brain. I think he’s absolutely right.”

Lectins Are Strongly Associated With Autoimmune Diseases of All Kinds

Since we just talked about carb-loading at least once or twice a week (once you’ve regained the ability to burn fat for fuel), it’s worth stressing that these ought to be healthy carbohydrates, and ideally lectin-free. While intermittent fasting and eating a ketogenic (high-fat, low-carb, moderate protein) diet will dramatically reduce your risk of chronic disease, lectins may still cause trouble. One of the primary issues is autoimmune diseases.

“One of the things I talk about in the book that really made me hyper-focused on lectins was a friend of mine who was a very early adopter of my first program. I call him Tony in the book. Tony had really bad vitiligo. That’s … where the [skin] pigmentation is lost. Vitiligo is an autoimmune disease.

What happens is we attack the pigment-forming cells in our skin called melanocytes. Melanocytes are actually modified neural cells. They migrate from the neural crest to our skin in embryonic development. When Tony started my program, a few months later, he came up to visit me. He said, ‘You’re not going to believe this. My vitiligo is gone.’ I’m looking at him and I’m going, ‘Wow. That’s impressive.’

He said, ‘How did that happen?’ I could have said, ‘Well, this is a very anti-inflammatory diet. It’s high in antioxidants.’ But because I’m a researcher, I said, ‘No. That’s too simple.’ I said, ‘Melanocytes. Neural Cells. What’s the target of lectins in insects? Neural cells! Could it be that lectins are why [his body is] attacking his neural cells? What I’ve done is I’ve removed lectins from his diet.’

I lost track of him for a number of years. I was on a health panel in New York City two years ago. I saw him and he’s covered with vitiligo again. I said, ‘What happened?’ He says, ‘You know. I fell off [the diet]. I really need to get back on.’ I said, ‘This is a great experiment. Come on. Here’s the list. Go for it.’

We were just on a panel at Harvard two months ago. He’s chairing the panel. He says, ‘I’ve got to show you — everybody — the vitiligo’s gone because I took lectins back out of my diet. It sounds silly but here’s the proof.'”

Molecular Mimicry

One way by which lectins cause harm is through molecular mimicry. They resemble proteins in the thyroid gland, in your joint spaces and in nerves. They mimic myelin sheath proteins.

The reason why lectins will in one person cause vitiligo or psoriasis, and in another attack the thyroid or cause rheumatoid arthritis, is still unknown. What is known is that one of the underlying factors in all of these disease processes is the penetration of the gut wall by lectins and their co-travelers, lipopolysaccharides (LPSs), also known as endotoxins, which tend to elicit very strong immune responses.

“One of the things I found in all my autoimmune patients is they had profoundly low levels of vitamin D … Interestingly, when you finally seal the gut … all of a sudden, their vitamin D levels went sky high and I could back down on the dosage.

Vitamin D is essential to tell the stem cells at the bottom of the crypts in the villi to grow and divide. Without vitamin D stimulating them, they just sit there and don’t repair the gut. I think plants are so intelligent, it’s shocking. I think one of the plant strategies is that if you have low vitamin D, because you can’t absorb it, then you can’t repair your gut. You’re a horrible predator. You won’t reproduce. You won’t walk.

Vitamin D is really one of the keys to autoimmune disease. Lectins are the other key. I’ve been blessed by knowing thousands of autoimmune patients who I call “canaries,” because they react almost instantaneously to lectins. It’s interesting. Everybody has their own certain lectin or lectins that they really react to.

This morning I had a woman who has rheumatoid arthritis. Her rheumatoid markers or anti-CCP3 markers have gone up. Her IL-17 had gone up. I said, ‘All right. What are you doing? What’s going on?’ She said, ‘No, no. I’m perfect. I know your list backwards and forwards.’ I said, ‘No. There’s something.'”

A Sample Case History of Crohn’s Disease

As it turns out, she’s been eating raw (unpeeled) almonds, and almond peels contain lectins. Another patient’s markers went up after going on a cashew binge, forgetting that cashews are an American bean and hence high in lectins. The answer for autoimmune patients, Gundry says, is to remove lectins from the diet and add vitamin D, which together will help “heal and seal” the gut, thereby preventing the autoimmune response.

“I mention a young woman who has Crohn’s disease in the book. Her well-meaning doctor at the Mayo Clinic told her that food had nothing to do with Crohn’s disease. She had been cured of Crohn’s disease with my program. He told her it was the placebo effect. We still laugh at that one. She ate a couple of Christmas cookies after she got off the phone with him.

Of course, it was like throwing a bomb in her stomach. She had horrible cramps and diarrhea. We skyped and she said, ‘Why don’t doctors see this?’ Like I talk about in the book, we can’t see unless our eyes are open …

I was lucky enough that when I met the guy who changed my life, Big Ed, who cleaned out his arteries with diet and supplements, [I had] my eyes open. I said, ‘This is not chance. How did [he] do this?’ Luckily, because of my evolutionary background, I was able to piece it together.”

Which Foods Have the Most Problematic Lectins?

Lectins are found in many of our most cherished foods, such as: 2,3

Potatoes Eggplants Tomatoes Peppers Goji berries Lima beans
Cashews Peanuts Sunflower seeds Chia seeds Pumpkin seeds Kidney beans
Squash Corn Quinoa Soybeans Wheat Lentils

Another common lectin is the A1 casein protein, found in most of today’s dairy cows. I’ve talked a lot about the benefits of raw milk on my site. The devil’s in the details however, and aside from being high in sugar, even raw dairy may cause problems if it has A1 casein.

“Casein A2 is the normal protein in milk, besides whey. It’s present in sheep, goats and water buffalos. But, most of the cows in the world are now casein A1 producers. They make a lectin-like protein called casein A1, which is metabolized in our gut to make beta-casomorphin, which is a very interesting thing. They can attach to the beta cell of the pancreas and incite an autoimmune attack on the pancreas.

I and others are pretty convinced that [many cases] of Type 1 juvenile diabetics is because of the casein A1 in milk. I’ve been convinced through the years that not only is it the problem, but people who think they’re lactose intolerant or that milk gives them mucus, it’s the casein A1 … Raw milk is great, as long as it came from the right cow … [Some] Jerseys are A1 and [some are] A2. Holsteins are A1.”

More and more people are now starting to recognize this, and there are even grassroots movements pushing for A2 milk in California and Ohio. Jeni’s Ice Cream gets all her milk from Snowville Creamery, which is an A2 farm. “I’ve actually talked to those people. They get it,” Gundry says. There have even been attempts to introduce A2 milk on a larger scale, but each attempt has been crushed by the American Dairy Council, for obvious reasons.

Wheat — Going Beyond Gluten

Wheat germ agglutinin (WGA) is another problematic lectin, found in wheat. Compared to WGA, gluten is a minor problem. According to Gundry, WGA is one of the most efficient ways to induce heart disease in experimental animals. WGA binds to insulin receptor sites. Normally, a normal hormone will dock on a receptor site, give its information and then release. Pseudo hormones like WGA, on the other hand, dock on the receptor permanently. Gundry explains:

“If they hit the insulin receptor on a fat cell, they turn on lipoprotein lipase and pump sugar into the fat cell, turning it into fat constantly. In muscle cells, the exact opposite happens.

They’ll attach to the insulin receptor in the muscle cell [and] block insulin from delivering sugar into the cell. I see so many long-distance runners who are carboholics, who look like concentration camp survivors because they’re really cachectic and sarcopenic because they block the insulin receptors in their muscles …

The lectins, like WGA and galactans in beans are miraculous ways of making us store fat … [T]he only way we’ve ever been able to fatten an animal for slaughter is to give them grains, beans and some antibiotics. If that’s how we fatten animals, that’s how we fatten us. It works really well.”

Not All Bread Is the Same

If you’ve ever traveled to Europe, you may have indulged in some bread and noticed you didn’t experience the same type of problems you have when eating bread in the U.S. The reason for this is because the lectins are removed when you use traditional methods of raising bread, which is still popular in Europe.

“Europe [has] always used traditional methods raising bread. They use yeast or sourdough. Yeast and bacteria are actually pretty good at breaking down the gluten molecule and other lectins,” Gundry explains.

Europe also does not permit the use of glyphosate to desiccate wheat, which has become common practice in the U.S. Glyphosate is also used on many conventional grains, including beans and flax, so it’s in the animal meats we eat, it’s in our baked goods, and even in wine produced in the U.S. According to Gundry, glyphosate potentiates gluten to people who are not even gluten-sensitive, and interferes with your liver’s ability to manufacture the active form of vitamin D.

Glyphosate also chelates important minerals, disrupts the shikimate pathway, decimates your microbiome and increases leaky gut, which allows more of the LPSs into your bloodstream. Since it works synergistically with the lectins, it really delivers a double-whammy.

“[Glyphosate] hits cytochrome P450. It’s one of the reasons the Europeans are so far [ahead] on health,” Gundry says. “It’s one of the reasons why so many of my patients can go to Europe, eat their traditional diet and think they’re cured and now they can start eating bread. They come back and eat a piece of bread and, bam — the whole thing starts all over again.”

On Vegetarianism and Other Diets

As mentioned, Gundry was a professor at Loma Linda University, a Seventh Day Adventist facility. Seventh Day Adventists are typically vegetarians, and while not an Adventist, Gundry did eat a vegetarian diet for about 15 years during his time there.

“I’ve never been sicker in my life. I used to weigh 228 pounds despite running 30 miles a week and running half marathons on the weekend and going to the gym one hour every day, wondering why I had high blood pressure, prediabetes and heart disease … Quite frankly, we have a fabulous orthopedic department at Loma Linda, because grains are pretty doggone mischievous for that.

Through the years, I’ve been good friends with the head of the Adventist Health Studies, a cardiologist. One of the things I’ve learned from following the Adventists and following Gary Fraser is that … certain animal proteins do contribute to aging. In the Adventist health study, the vegan Adventists have the longest life span. Behind them are the lacto-ovo vegetarians, then behind them are the pescetarians. Then finally, there are the real cheaters who eat chicken …

It is interesting that the longest living of the Adventists, who are very long-living, are the vegans. I take care of a lot of vegans because of my association with Loma Linda. As a general rule, the vegans are some of the unhealthiest people I have met. The reason is they’re grain- and bean-itarians. They are not vegetable eaters.

I have nothing against a high vegetable diet … The other thing we see in the vegans is they somehow think they will convert short-chain omega-3 fats into EPA, the long-chain omega-3 fats. They absolutely and positively do not.

Our brain is about 70 percent fat; 50 percent of that fat is DHA. There are beautiful longitudinal studies showing people with the highest omega-3 index have the largest brains as they age, and the largest areas of memory in the hippocampus. People with the lowest levels of omega-3 index have the most shrunken brains and the smallest areas of memory. Vegans have no excuse anymore. There’s algae-based DHA.”

Fruit and Berries — Seasonal Treats

Gundry’s first rule is that what you stop eating is more important than what you start eating. “It’s absolutely true,” he says. “If you take away certain foods, you’ll be amazed [to find] that it’s certain foods that are the troublemakers.” His second rule is, take care of your gut microbiome. Rule No. 3 is “fruit might be as good as candy.” While he doesn’t expound on the importance of burning fat for fuel in his book, that’s really part of the equation. Once you’re able to burn fat, fruit can be a healthy carbohydrate to add once or twice a week.

“Exactly. I think part of the problem is the vast majority of Americans are insulin-resistant. One of the things that people should realize is that the modern fruit has been bred for sugar content … One of the things I ask people to do initially is give fruit the boot.

Fructose is a major toxin. We take fructose directly to our liver and detoxify it into triglycerides and uric acid. It always amazes me the number of people with gout who consume more concentrated fruit, like wine or beer. Beer is one of the underlying reasons that they have gout.

The other thing people should realize is that fructose is a direct renal toxin. The more fructose I can get out of people, the better. Having said that, once you get to a point where you have metabolic flexibility, I think things like berries are probably one of the best ways to carbohydrate load on the day you decided to do that … Sweet potatoes are great as well, [and] I’m a big fan of taro root.

Years ago [in June] … my wife and I were at a Santa Barbara farmers market. I was taking these gorgeous organic peaches and putting them into my bag. She says, ‘Hey, wait a minute. Aren’t you the guy who says give fruit the boot?’ I said, ‘Yeah, yeah. But it’s June and it’s time to eat fruit.’ She says, ‘OK, smart guy. Let’s do this. This summer, we’re going to give up fruit to see what happens’ …

So, we gave up fruit for one summer. We didn’t change anything else in our diet. My wife lost 6 pounds and I lost 8 pounds. It brought home to me that, again, our ancestors and the reason we have two-thirds of our tongue devoted to sweet taste is we are great fruit predators. Fruit was only available once a year. We utilized that fruit to gain weight for the winter … [Now] we can have it 365 days a year, but that’s not normal. So, always keep that in mind.”


Connie: Eat gluten free, avoiding refined and processed foods.

Use Distributorship ID #: USW9578356 when ordering at:

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Fasting, sun bathing ,Vit C, Lysine, turmeric, green tea, carrots and raw food diet to reduce tumor size

This section is from the book “The Hygienic System: Fasting And Sun Bathing“, by Herbert M. Shelton. Also available from Amazon: The Hygienic System Vol III Fasting and Sun Bathing.

The Autolytic Disintegration Of Tumors

Trall asserted that all abnormal growths possess a lower grade of vitality than normal growths, hence are easier to destroy. I think it may be equally true that they do not command the support of the organism as do normal growths, as they are lacking in nerve and blood supply. This lack of support makes them the ready victims of the autolytic processes of the body. It is generally held by men with wide experience with the fast that abnormal tissues are broken down and eliminated more rapidly than normal tissue during periods of abstinence. Physiologists have studied the process of autolysis, although they have suggested no practical use that may be made of it save that of employing it to reduce weight. It now remains for physiologists to learn that by means of rigidly controlled autolysis, the body is able to digest tumors and utilize the proteins and other food elements contained in them to nourish its vital tissues. Why have they not investigated this vitally important subject? The facts have been before the world for more than a hundred years.

More than a hundred years ago Sylvester Graham wrote: “It is a general law of the vital economy, that when, by any means, the general function of decomposition exceeds that of composition or nutrition, the decomposing absorbents always first lay hold of and remove those substances which are of least use to the economy; and hence, all morbid accumulations, such as wens, tumors, abscesses, etc., are rapidly diminished and often wholly removed under severe and protracted abstinence or fasting.”–Science of Life, pp. 194-195.

The process of autolysis may be put to great practical use and may be made to serve in getting rid of tumors and other growths. To fully understand this, it is necessary for the reader to know that tumors are made up of flesh and blood and bone. There are many names for the different kinds of tumors, but the names all indicate the kind of tissue of which the tumor is composed. For example, an osteoma is made up of bone tissue; a myoma is composed of muscular tissue; a neuroma is constituted of nerve tissue; a lipoma consists of fatty tissue; a fibroma is composed of fibrous tissues; an epithelioma is composed of epithelial tissue, etc. Growths of this nature are known, technically, as neoplasms (new growth) to distinguish them from mere swellings or enlargements. A large lump in the breast may be nothing more than an enlarged lymphatic gland, or an enlarged mammary gland. Such an enlarged gland may be very painful, but it is no neoplasm.

Tumors being composed of tissues, the same kinds of tissues as the other structures of the body, are susceptible of autolytic disintegration, the same as normal tissue, and do, as a matter of experience, undergo dissolution and absorption under a variety of circumstances, but especially during a fast. The reader who can understand how fasting reduces the amount of fat on the body and how it reduces the size of the muscles, can also understand how it will reduce the size of a tumor, or cause it to disappear altogether. He needs, then, only to realize that the process of disintegrating (autolyzing) the tumor takes place much more rapidly than it does in the normal tissues.

In his Notes On Tumors, a work for students of pathology, Francis Carter Wood says: “In a very small proportion of human malignant tumors spontaneous disappearance for longer or shorter periods has been noted. The greater number of such disappearances has followed incomplete surgical removal of the tumor; they have occurred next in order of frequency during some acute febrile process, and less frequently in connection with some profound alteration of the metabolic processes of the organism, such as extreme cachexia, artificial menopause, or the puerperium.”

No more profound change in metabolism is possible than that produced by fasting and the change is of a character best suited to bring about the autolysis of a tumor, malignant or otherwise.

The conditions Dr. Wood mentions as causing spontaneous disappearance of tumors are, for the most part, “accidents” and are not within the range of voluntary control. Fasting, on the other hand, may be instituted and carried out under control and at any time desired. It is the rule that operations are followed by increased growth in the tumor. Spontaneous disappearance following incomplete removal is rare. The same may be said for extreme cachexia and artificial menopause. In fevers we have rapid autolysis in many tissues of the body and much reparative work going on, but we cannot develop a fever at will. Pregnancy and childbirth occasion many profound changes in the body, but they are certainly not to be recommended to sick women as cures for their tumors. Even if this were desirable, it would be a hit-or-miss process. The effects of fasting are certain. There is nothing hit-or-miss about the process. It works always in the same general direction.

Fever is a curative process and does help to remove the cause of the tumor. None of Dr. Wood’s other causes of spontaneous disappearance assist in removing the cause of tumors. Fasting does assist greatly in the removal of such cause.

During a fast the accumulations of superfluous tissues are overhauled and analyzed; the available component parts are turned over to the department of nutrition to be utilized in nourishing the essential tissues; the refuse is thoroughly and permanently removed.

Due to a variety of circumstances, some known, others unknown, the rate of absorption of tumors in fasting individuals varies. The general condition of the patient, the amount of surplus contained in is body, the kind of tumor, the hardness or softness of the tumor, the location of the tumor and the age of the patient are all known to influence the rate of tumor absorption. Let me cite two extreme cases to show the wide range of variation in this respect.

A woman, under forty, had a uterine fibroid about the size of an average grapefruit. It was completely absorbed in twenty-eight days of total abstinence from all food but water. This was an unusually rapid rate of absorption. Another case is that of a similar tumor in a woman of about the same age. In this case the growth was about the size of a goose egg. One fast of twenty-one days reduced the tumor to the size of an English walnut. The fast was broken due to the return of hunger. Another fast a few weeks subsequent, of seventeen days, was required to complete the absorption of the tumor. This was an unusually slow rate of tumor-absorption.

Tumor-like lumps in female breasts ranging from the size of a pea to that of a goose egg will disappear in from three days to as many weeks. Here is a remarkable case of this kind that will prove both interesting and instructive to the reader. A young lady, age 21, had a large, hard lump–a little smaller than a billiard ball–in her right breast. For four months it had caused her considerable pain. Finally she consulted a physician who diagnosed the condition, cancer, and urged immediate removal. She went to another, and another and still another physician, and each made the same diagnosis and each urged immediate removal. Instead of resorting to surgery the young lady resorted to fasting and in exactly three days without food, the “cancer” and all its attendant pain were gone. There has been no recurrence after twenty-three years and I think that we are justified in considering the condition remedied.

Hundreds of such occurrences under fasting have convinced me that many “tumors” and “cancers” are removed by surgeons that are not tumors or cancers. They cause me to be very skeptical of the statistics issued to show that early operation prevents or cures cancer.

Let me cite a comparatively recent instance from my own practice. A manufacturer brought his wife to me from Los Angeles. A growth in one of her breasts had caused her to consult two or three physicians in that city. Each of them had insisted upon the immediate removal of her breast. I placed her upon a fast which was continued for thirty days. At the end of the fast, the tumor, which was about the size of an English walnut at its beginning, had been reduced to the size of a pea. In less than a month on a vegetable and fruit diet this small remainder disappeared.

Subsequently the woman gave birth to two children at about two year intervals. She nursed each child for two years during which nursing periods the formerly tumurous breast functioned well. The health and vigor of the boys presented unequivocal evidence of the quality of the mother‘s milk. Was this not better than removal of the breast? Was this an exceptional case? By no means. I see them regularly. Such cases are seen daily in institutions in various parts of the world where fasting is employed.

. The removal of tumors by autolysis has several advantages over their surgical removal. Surgery is always dangerous; autolysis is a physiological process and carries no danger. Surgery always lowers vitality and thus adds to the metabolic perversion that is back of the tumor. Fasting, by which autolysis of tumors is accelerated, normalizes nutrition and permits the elimination of accumulated toxins, thus helping to remove the cause of the tumor. After surgical removal tumors tend to recur. After their autolytic removal, there is little tendency to recurrence. Tumors often recur in malignant form after their operative removal. The tendency to malignancy is removal by fasting.

John W. Armstrong (England) says: “I have seen lumps in female breasts treated to fast, some of them after diagnosis by ‘experts,’ the bulk after self-diagnosis and to disappear, on water only, in from four to twenty days.”

Bernarr Macfadden says: “My experience of fasting has shown me beyond all possible doubt that a foreign growth of any kind can be absorbed into the circulation by simply compelling the body to use every unnecessary element contained within it for food. When a foreign growth has become hardened, sometimes one long fast will not accomplish the result, but where they are soft, the fast will usually cause them to be absorbed.”

A small tumorous growth which had existed for more than twenty years was absorbed during Mr. Pearson’s longest fast and did not return thereafter. Dr. Hazzard records the recovery, during a fifty-five days’ fast, of a case diagnosed by physicians as cancer of the stomach. Tilden, Weger, Rabagliati and many others record many such cases.

I have seen repeated instances of the absorption of tumors in my own patients. I had one complete recovery in the case of a uterine cancer during a thirty days’ fast. I have seen numerous small tumors completely absorbed and large ones greatly reduced in size.

In Europe and America, literally thousands of tumors have been autolyzed during the past fifty years, and the effectiveness of the method is beyond doubt. I can give no definite information about bone tumors and nerve tumors; but, since these are subject to the same laws of nutrition as all other tumors, I am disposed to think that they may be autolyzed as effectively as other tumors.

In my own experience I have seen numerous fibroid tumors of the uterus and breast, lipomas in various parts of the body, a few epitheliomas, a whole group of myomas and a number of tumors that were apparently early cancer autolyzed and absorbed while the patient fasted. I have seen many warts disappear during fasting and I have seen many warts on which the fasting process seemed to have no effect. I have never seen a mole affected by the fasting process. I have seen a number of cysts completely destroyed by fasting and others that were merely reduced in size. It will be recalled that Graham mentions having seen cysts (wens) absorbed during fasting.

It is certain that the autolyzing process has its limitations. For example, a tumor that has been permitted to grow to enormous size cannot be autolyzed in one fast. Indeed, many of them are so large that several long fasts during the course of two years or more, with a rigid feeding schedule between fasts, would be required to break them down and absorb them, if, indeed, it could be done. There was a school in Chicago some years ago that taught that “the normal tissue may be consumed before the morbid tissues are used up,” in fasting. While this school did not confine this statement to tumors, there are few conditions in which this can be a fact, and in large tumors it may be so. Aside from large tumors, it is hardly probable that this is so in any recoverable cases. Only in rare instances, where the amount of morbid tissue is very great, and these are probably all irremediable, can this occur.

In general, good tissue is not used up as fast as bad and the tumor will “starve” before the body. Except where it is very large, we may be sure that in all cases, hunger will return before any damage is done to the vital tissues. In more than one case of cancer, where opiates had been used to relieve pain, I have seen three or four days’ fasting bring relief.

One other limitation must be noted; namely, tumors that are so situated that they dam-up the lymph stream will continue to grow (feeding upon the excess of lymph behind them) despite fasting.

In cases where complete absorption is not obtained, the tumor is sufficiently reduced in size not to constitute a menace. Thereafter proper living will prevent added growth. Indeed, we have seen a number of cases where a further decrease in size followed right living subsequent to fasting.

Read more: http://chestofbooks.com/health/natural-cure/The-Hygienic-System-Fasting-and-Sun-Bathing/The-Autolytic-Disintegration-Of-Tumors.html#ixzz2YTcTUK00

Note: Do use sunglasses and face sunscreen. Avoid high UV during 11am to 2pm.

DOCTOR YOURSELF NEWS: Dr. Holick, What are some of the research-based benefits of more vitamin D?

DR. MICHAEL HOLICK: You can reduce cancer risk by 30 to 50% by increasing vitamin D in the diet, or by sensible sun exposure. We gave mice colon cancer, and followed them for 20 days. Tumor growth was markedly reduced simply by having vitamin D in the diet. There was a 40% reduction in tumor size. And, casual sun exposure actually decreases your risk of melanoma. In Finland, back in the 1960’s, children that received 2,000 IU of vitamin D each day reduced their risk of getting Type 1 diabetes by 80%. Every tissue and every cell in your body has a receptor for vitamin D. Every tissue and every cell of your body requires vitamin D to function properly.

DY NEWS: Exactly how much vitamin D, and how much sun, do we need?

HOLICK: Vitamin D deficiency is less than 400 IU per day along with no sun exposure. If you take 400 IU daily, you would still have an inadequate amount of vitamin D. Humans need 1,000 IU each day, or to be exposed to sunlight. Five to ten minutes, arms and legs, three times a week, is adequate.

DY NEWS: What is you opinion of the official US RDA/DRI recommendations?

HOLICK: I was on the committee that set them (National Academy of Sciences, Panel on Calcium and Related Nutrients, 1996-1997). We have made some progress in increasing the recommendations. They are now 200 IU/day for children and adults up to age 50; 400 IU for adults over 50; and 600 IU/day for adults over 70.

DY NEWS: Would you have liked to have set the recommendations higher?

HOLICK: Yes. At the time, we were obligated to base our recommendations on the published literature before 1997. Based on new evidence, I think infants, up to one year of age, need 400 IU/day. Canada recommends 400 IU already. Then, from age one, and all through adulthood, I’d recommend 1,000 IU/day. Everyone needs 1,000 IU of vitamin D3 each day.

DY NEWS: And how are we doing?

HOLICK: In Boston, 50% of adolescent boys and girls are vitamin D deficient. 70% of moms and 80% their babies are vitamin D deficient at birth. These infants have no vitamin D stores, and the moms have none to give them.

DY NEWS: And that means rickets?

HOLICK: Rickets is only the tip of the vitamin D deficiency iceberg. If you are vitamin D deficient in childhood, you are 2.4 times more likely to develop Type 1 diabetes.

DY NEWS: And for people of color?

HOLICK: Skin pigment is a natural sunscreen. African American children require two to three times as much sun exposure, without sunscreen, to satisfy their requirement for vitamin D.

DY NEWS: What are the consequences of not getting enough sunlight?

HOLICK: If you live above 35 degrees north latitude, you are twice as likely to develop multiple sclerosis. Living in higher latitudes also means higher risk for Crohn’s disease, rheumatoid arthritis, and high blood pressure.

DY NEWS: What are some examples of “high latitude” cities?

HOLICK: Anything above Atlanta, Georgia.

DY NEWS: I’ve been to Atlanta. To a Yankee like me, that’s pretty far south. Now you know I’m going to quote you on this. . .

HOLICK: That’s fine. Anywhere above about 35 or 37 degrees latitude, that is, anywhere north of Atlanta, Georgia, you basically cannot make vitamin D in your skin during the wintertime.

DY NEWS: And in the summer, should we tan?

HOLICK: I do not believe in tanning.

DY NEWS: That seems a rather moderate position.

HOLICK: I was fired from my position as Professor of Dermatology at Boston University Medical Center because I have been promoting sensible sun exposure, and had written the book, “The UV Advantage.” I had held that position for nearly ten years.

DY NEWS: Who fired you, and when?

HOLICK: Dr. Barbara Gilchrest, BU Medical Center Chief of Dermatology, in February 2004. She and I remain personal friends. I’m still full Professor of Medicine, Physiology, and Biophysics, and have been for 20 years. Dr. Gilchrest has been quoted as saying that to suggest that vitamin D deficiency is a significant health problem is “weak and absurd.” She has also been quoted as saying that linking vitamin D deficiency to medical illness is “schlock science.” I have never heard her rebut those statements.

DY NEWS: Were you fired entirely because of your stance on vitamin D and for no other reason?

HOLICK: No question about it. The American Academy of Dermatology is very uncomfortable about sensible sun exposure, and anyone who recommends it.

DY NEWS: You’re telling us that the American Academy of Dermatology does not believe in sensible sun exposure?

HOLICK: That is correct. They are believers in abstinence from all sun exposure. They have been pretty firm about this. In May 2004, three months after I was fired, I was asked to defend myself in front of all the staff.

DY NEWS: How successful was it?

HOLICK: I got people’s attention.

DY NEWS: Who is most opposed to you?

HOLICK: The “unenlightened” dermatologists of the American Academy of Dermatology.

DY NEWS: Yet it seems to be particularly difficult to kill yourself with vitamin D.

HOLICK: True. One man took 1,000,000 IU of vitamin D per day, orally, for six months. Of course, he had the symptoms of severe vitamin D intoxication.

DY NEWS: But he lived to tell the tale?

HOLICK: Yes. His treatment was hydration (lots of water), and no more vitamin D or sunshine for a while. He’s perfectly happy and healthy. This was published in the New England Journal of Medicine. (Koutkia P, Chen TC, Holick MF. Vitamin D intoxication associated with an over-the-counter supplement. N Engl J Med. 2001 Jul 5;345(1):66-7.)

DY NEWS: How many people have died from vitamin D or other vitamins?

HOLICK: I have no experience of anyone dying from vitamin exposure. In thirty years, I’ve never seen it.

DY NEWS: And in the medical literature?

HOLICK: Not as far as I know.

DY NEWS: About how many people get too little vitamin D?

HOLICK: In the US and Canada, about 50%. About one billion people, worldwide, are vitamin D deficient. This is true even in sunny climates, because of lack of sun exposure.

DY NEWS: What advice do you wish to offer our readers?

HOLICK: The most important thing is to increase your vitamin D intake.

DY NEWS: What’s yours?

HOLICK: I take 1,100 IU of vitamin D every day.

Shrinking tumors

In many cases, it is critical to the survival of a cancer patient to shrink their tumors. However, in many other cases it is not life-threatening to leave a tumor alone and concentrate on stopping the spreading of the cancer.

“Modern medicine,” with its firm grasp of the art of making huge amounts of profits, has totally brainwashed the public into thinking that the size of every tumor is important in a cancer treatment. In many cases shrinking the tumor is critical!! However, orthodox medicine frequently shrinks tumors which are irrelevant to the survival of the patient. Here is a quote by Dr. Philip Binzel, M.D.:

  • “When a patient is found to have a tumor, the only thing the      doctor discusses with that patient is what he intends to do about the      tumor. If a patient with a tumor is receiving radiation or chemotherapy,      the only question that is asked is, “How is the tumor doing?” No      one ever asks how the patient is doing. In my medical training, I remember      well seeing patients who were getting radiation and/or chemotherapy. The      tumor would get smaller and smaller, but the patient would be getting      sicker and sicker. At autopsy we would hear, “Isn’t that marvelous!      The tumor is gone!” Yes, it was, but so was the patient. How many      millions of times are we going to have to repeat these scenarios before we      realize that we are treating the wrong thing?

In primary cancer, with only a few exceptions, the tumor is neither health-endangering nor life-threatening. I am going to repeat that statement. In primary cancer, with few exceptions, the tumor is neither health-endangering nor life-threatening. What is health-endangering and life-threatening is the spread of that disease through the rest of the body.

There is nothing in surgery that will prevent the spread of cancer. There is nothing in radiation that will prevent the spread of the disease. There is nothing in chemotherapy that will prevent the spread of the disease. How do we know? Just look at the statistics! There is a statistic known as “survival time.” Survival time is defined as that interval of time between when the diagnosis of cancer is first made in a given patient and when that patient dies from his disease.

In the past fifty years, tremendous progress has been made in the early diagnosis of cancer. In that period of time, tremendous progress had been made in the surgical ability to remove tumors. Tremendous progress has been made in the use of radiation and chemotherapy in their ability to shrink or destroy tumors. But, the survival time of the cancer patient today is no greater than it was fifty years ago. What does this mean? It obviously means that we are treating the wrong thing!”


Philip Binzel, M.D., Alive and Well, Chapter 14

While there are some alternative cancer treatments that do shrink tumors, and this article is about those products, the focus on alternative cancer treatments is generally on targeting and killing cancer cells, or reverting cancer cells into becoming normal cells. Shrinking tumors is generally secondary to alternative cancer treatments. But again, in some cases the tumor is life-threatening.

For example, in some cases the tumor is pressing on a vital organ, is causing pain, is obstructing the flow of fluids, or for some other reason needs to be eliminated from the body. If the problem is life-threatening the solution is frequently solved with surgery because of the urgency of the situation.

It is important to understand that there is no alternative cancer treatment that is guaranteed to shrink tumors significantly in a short amount of time. Tumors are very complex and there is nothing that will work every time.

Not all alternative cancer treatments that shrink tumors are listed here, but the major treatments that have a history of shrinking tumors are listed here.

When the size of a tumor is critical, it can be assumed you do not want to swell the tumor any more than it has already swollen. Most alternative cancer treatments will cause a tumor to swell temporarily, then it may start to shrink.



DMSO Protocols

DMSO is an amazing molecule as it can pass through the skin like it wasn’t even there. And it can carry things with it or “meet up” with things taken orally – at the site of the tumor.

There are two options for using DMSO. First, is the Overnight Cure For Cancer, which is a DMSO and chlorine dioxide protocol. DMSO and chlorine dioxide actually bind to each other. DMSO carries the chlorine dioxide with it through the skin (it is taken transdermally) and heads for the cancer cells.

This article discusses this protocol, but remember that DMSO should be put on the skin directly on top of the tumor or on the skin directly above the tumor:
Overnight Cure For Cancer

The second way to use DMSO is with baking soda. This has done very, very well against external tumors where you can put the DMSO and baking soda directly on the tumor. Plus, this protocol is dirt cheap for those on a limited budget, but so is the Overnight Cure For
Cancer protocol.

Actually, the baking soda protocol is ADDING baking soda to the DMSO/chlorine dioxide protocol just mentioned!!

No more than 1 TEAspoon a day, total, of baking soda, should be taken by an adult. Baking soda should only be added to the Overnight Cure For Cancer for a maximum of six weeks.

This protocol is to mix 2 TEAspoon of DMSO with 1/3 TEAspoon of baking soda, THREE times a day. This also means DMSO and chlorine dioxide is used three times a day!!

This combination will generally be taken externally, either directly on the tumor or on the skin above where the tumor is.


Amazon Tonic III, Botanical Support, Etc. (i.e. Herb Healers)

Amazon Tonic III is an herbal product which has been around for many years under the name of “Cansema.” It has proven itself over the course of many years to be superb at shrinking tumors.

The term “Cansema” is now a generic name so do not use any product with the “Cansema” name. The “Amazon Tonic III” is the product of one of the pioneers in developing Cansema.

For Amazon Tonic III, here are the instructions:
1) Shake the bottle well before using it (do this each time it is used),
2) Refrigerate after opening,
3) Start the dose at 1/2 TEAspoon, 3 times a day ON A FULL STOMACH,
4) Build up to 1 TEAspoon, 3 times a day ON A FULL STOMACH,
5) You can mix a small amount of juice with this product because it tastes bad.

Other available products include “Botanical Support.”

Work with the vendor for dealing with your situation. Here is the website to obtain these, and other, herbal products:
Herb Healers Website


Cesium Chloride

If the tumor is not life threatening, should it swell temporarily (e.g. for a couple of weeks), the Cesium Chloride / DMSO Protocol is a good choice. This is one of the best and fastest treatments to shrink tumors. Tumor masses can start to shrink within two weeks. However, before the tumor starts to shrink, as with most alternative cancer treatments that shrink tumors, the tumor will actually get larger for a short time (unless the patient is on chemotherapy). Thus, it must be known whether temporary swelling will put the cancer patient at risk.

Cesium Chloride and the Cellect-Budwig (to be discussed next) are similar enough protocols that they should not be taken at the same time, or even one after the other.

If the cancer patient is currently on chemotherapy, the combination of either of these protocols, combined with chemotherapy, is a very good choice. Both seem to be synergistic with chemotherapy.

See the left side-bar for links to these two protocols.


Budwig Diet and/or Cellect-Budwig

The Cellect-Budwig Protocol contains the Budwig Diet, which consists of converting oil soluble omega 3 into water soluble omega 3. This treatment is also part of the Bill Henderson Protocol, which will be mentioned in a moment.

Dr. Johanna Budwig claimed that tumors were created specifically because of a lack of electron-rich molecules on the surface of cell walls. Her protocol was excellent at shrinking tumors quickly.

In addition to the Budwig Diet, the product Cellect can also shrink tumors. The Budwig Diet and Cellect are very synergistic. Cellect contains, among many other products, shark cartilage, which is known to shrink tumors.

Another part of the Cellect-Budwig protocol is vegetable juicing. Heavy vegetable juicing, especially carrot juice, beet juice, cabbage juice, broccoli juice and other anti-cancer juices, are known to help shrink tumors.


The Kelmun Protocol

For those on a budget, the Kelmun Protocol (baking soda and maple syrup) has done very well at shrinking tumors. It is both a highly alkaline protocol and it contains a “trojan horse” (maple syrup) to allow the baking soda to target cancer cells.

It should be combined with juicing (e.g. carrot juice with a tablespoon of beet juice) and “green drinks” and hopefully an electromedicine protocol, such as the GB-4000 M.O.P.A.

Also see the “Dirt Cheap Protocol” for more ideas to add to the Kelmun Protocol:
Dirt Cheap Protocol

Also see the “Inexpensive Cancer Treatments” article, which is linked to on the left side-bar (the column of links on the left side of most pages).


Bill Henderson Protocol

The Bill Henderson Protocol is also a protocol which includes the Budwig Diet and it also does not cause any inflammation or swelling of tumors. It is based on the Johanna Budwig Diet of cottage cheese and flaxseed oil. The treatment also includes several other supplements and a special diet.

However, since the Bill Henderson Protocol is not specifically designed to shrink tumors, and since by definition, the patient is already in serious trouble, there are things that should be added to the Bill Henderson Protocol when shrinking tumors is important (adult dosages):
1) 10 grams of MSM a day to reduce swelling (must build-up to this dose):
How To Make MSM Water

2) Shark cartilage. A person should take three 750 mg. pills a day. See:
One of many vendors

3) Heavy vegetable juicing, especially carrot juice, beet juice, cabbage juice, broccoli juice and other anti-cancer juices. See:
Anti-Cancer Vegetable Juices

Here is a special article on the Bill Henderson Protocol, along with a link to Bill Henderson’s website, Beating Cancer Gently:
Bill Henderson Protocol and Link


Honey and Turmeric and Ginger [Everyone Should Use]

Honey is a Trojan Horse to get microbe-killing substances inside of cancer cells, to revert cancer cells into normal cells.

In two different studies, turmeric (aka curcumin) was the best substance to kill the specific microbe that causes cancer.

Every other day take 2 TEAspoons of honey with 1 TEAspoon of turmeric (or curcumin)
Every other day take 2 TEAspoons of honey with 1 TEAspoon of ginger
The Ginger and Turmeric are alternated every other day, thus every day you are taking one or the other.


Sister Mary Eymard Poydock, PhD Treatment [Everyone Should Use]

This treatment is the result of decades of cancer research by Sister Mary Eymard Poydock, Ph.D., director of cancer research and former professor of biology at Mereyhurst College in Erie, Pennsylvania.

Twenty years of painstaking work by Sister Eymard and her associates have indicated that a combination of vitamins C and B12 have a powerful effect against tumors.

Sister Eymard stated: “We’ve got it down to a point now where, if you do it according to the ‘recipe,’ it will work every time.”

The “recipe” is a mixture of vitamins C and B12 (in a ratio of one part B12 to two parts C) near the site of the tumor.

She also wanted to see if the C-B12 mixture would prolong the lives of animals already suffering from cancer. To find out, Sister Eymard and her colleagues injected the mixture near the cancerous growths of diseased mice for seven successive days.

Treated animals lived longer than those mice not given C and B12. In fact, all the treated mice outlived the control group. It appeared that the combination of C and B12 not only inhibits the growth of cancer cells, but also prolongs the lives of animals impregnated with cancer.

One rounded teaspoon contains 4 grams of absorbic acid and 700 mg of potassium ascorbate. As with all vitamin C products, keep this product out of the reach of children!! It can be very dangerous if very high doses are taken.

This is an excellent source of Vitamin B12 which is highly absorbed:
http://www.wonderlabs.com/sublingual-b12/


Laetrile

Laetrile is another natural product known to help shrink tumors. Vitamin B17, laetrile, and amygdalin are all names for the same natural molecule. Apricot seeds are very high in laetrile.

This protocol will not cause tumors to swell prior to shrinking.

Here is a link to the Cellect-Budwig protocol:
Cellect-Budwig Protocol


Zeolites

Zeolites are specific types of minerals which come from volcanoes. They are best known as chelating molecules, especially for mercury and other heavy minerals. But it turns out that they are also very good at shrinking tumors.

  • For centuries, the powdered forms of specific zeolites have been      used as traditional remedies throughout Asia to promote overall health and      well being. The story of the “volcanic rocks” has been passed      down from generation-to-generation as more and more people have experienced      its life-changing benefits.

Zeolites are natural volcanic minerals with an unique, complex crystalline structure. It’s honeycomb framework of cavities and channels (like cages) works at the cellular level trapping heavy metals and toxins. In fact, because it is one of the few negatively charged minerals in nature, zeolites act as magnets drawing toxins to it, capturing them in its cage and removing them from the body.”

Here is a vendor of Pure Body Extra Strength (spray), to chelate the blood, and Pure Body (liquid), to chelate the the colon:
My Touchstone Essentials

Of interest is that they have been shown in some cases to shrink tumors very quickly. There is not enough evidence to depend on them exclusively to shrink tumors, but they are so simple to take that I would strongly recommend they be added to any of the other treatments that shrink tumors.

See also:
Zeolite Article by an M.D. [Part 1]
Zeolite Article by an M.D. [Part 2]


AIS MAX For Life

This is the only alternative cancer treatment I have ever seen with curcumin in it. There is anecdotal evidence this product does shrink tumors.

Curcumin is actually a wonder substance against cancer. Big Pharma has tried to synthesize this natural molecule, which works quite well by itself, thank you.

Because the dosage of curcumin in this product is somewhat lower than I would like, it would be good to use curcumin spice on your foods whenever appropriate while using this product and as long as you have cancer.

As with zeolites, the evidence is not strong enough that it shrinks tumors (i.e. there is no “cure rate”) that it should be used by itself.

Here is the vendor:
AIS MAX For Life


Sweet Wormwood Herb

A third product (there is no article for this product on this website) that may also shrink tumors very quickly is the sweet wormwood herb. This is generally not used as a stand-alone treatment, however, it may be very effective at shrinking tumors. The vendor with the best reputation for sweet wormwood is Artemisinin:
Sweet Wormwood Vendor


Essaic Tea (Using herbs that have NOT been dehydrated)

Essaic Tea is one of the foremost alternative cancer treatments known to shrink tumors. As with all herb teas, however, its ability to work is heavily dependent on the quality of the growing, processing and storage of the herbs. Herbs that have been dehydrated are rarely of much use.

    • “After word of Caisse’s [the founder of Essiac Tea]       impressive results spread to the United States, a leading diagnostician       in Chicago introduced her to Dr. John Wolfer, director of the tumor clinic       at Northwestern University Medical School. In 1937, Wolfer arranged for       Rene to treat thirty terminal cancer patients under the direction of five       doctors. Rene commuted across the border to Chicago, carrying her bottles       of freshly prepared herbal brew. After supervising one and a half years       of Essiac therapy, the Chicago doctors concluded that the herbal mixture       prolonged life, shrank tumors, and relieved pain. ”
      http://www.healthy.net/asp/templates/article.asp?PageType=article&ID=2008

Carrot Juice and Raw Food Diet

Vitamin A has been shown in studies to shrink the size of tumors. The best way to take Vitamin A is with carrot juice:

    • “Yellow to Deep Orange (carotenoids [Beta-carotene is the       most famous] are a huge family of over 600 “colors” that make       Vitamin A and help to shrink tumors):”
      http://www.learningplaceonline.com/nutrition/food/ca-prevent/fruit-veg.htm
    • “Cancer growths and sores appear in practically every part       of the body and take a long time to heal. Since the body creates these       conditions, it is essential to eliminate the food which feeds their       development. From his long experience, Dr. Earp-Thomas was fully       convinced that when cooked food was eaten it permitted tumors and growths       to build within the body. Yet when living food was substituted, these       tumors and growths immediately began to shrink for lack of nourishment.       The most thrilling experience I can recall was to see cancer cells taken       from a human body and thriving on cooked food but unable to survive on       that same food when it was uncooked.”
      http://www.karlloren.com/diet/p66.htm
    • “There are numerous reports/claims that a diet consisting       almost entirely of a diversity of vegetables can cause cancer tumors to       shrink or even vanish. If these reports are correct, they make sense in       the context of the theory that is presented here. I am postulating       anaerobic metabolism and thus cancer can be caused by the blockage of the       aerobic process at any point in the Kreb’s cycle or the respiratory       chain. There are numerous chemical steps that are involved in sequence       and if any of them is blocked you have the potential of arresting the       entire aerobic process. Each of these steps are catalyzed by specific       enzymes and regulated by other enzymes. If any of these are missing,       aerobic metabolism could stop.”
      http://www.krysalis.net/cancer.htm

Ellagic Acid

    • “Multiple studies have discovered that phytonutrients found       in raspberries can protect us from cancer and can even shrink some types       of cancer tumors. These substances can also act as an antibacterial and       as an antiviral agent. Does this sound too good to be true? One       particular substance found in this natural “medicine chest”, is       a series of compounds called ellagitannins. The highest levels are found       in raspberries, but the ellagitannins are also in certain types of       grapes, strawberries, blackberries, blueberries and some nuts too. Recent       work (2001), published by Dr. Gary Stoner at Ohio State University, showed       that components in the seeds and berry, but particularly ellagitannins,       inhibited the initiation and promotion/progression stages of esophageal       cancer. This is an extremely important finding, considering the potential       benefits.”
      http://www.smdi.org/new_page_3.htm
    • “The studies found in the left column [of this web page]       were compiled by Susan Thorpe-Vargas Ph.D. and represent a summation of       EA’s known medical properties:
      EA is an anticancer agent – it protects DNA from mutation.
      EA shrinks tumors within 72 hours if the cancer is not caused by a       mutation in the p53 or WAF 1/p21 genes.
      EA is a natural phenolic antioxidant.
      EA exerts actions that are both anti-bacterial and an anti-viral.
      EA appears to inhibit liver fibrosis.
      EA is cardioprotective and may prove useful in the treatment of       myocarditis.”
      http://health.centreforce.com/health/ellagic.html

Coenzyme Q10 (CoQ10)

    • “In one breast cancer study, women were given 390 mgs per       day in divided doses along with supplements. The result was that tumors       shrank. A maintenance dose of 100 mgs was considered adequate.”
      http://www.fightingcancer.com/supplements.htm

Actually, the recommended dose of CoQ10 is at least 390 mgs.


Aloe Arborescens (i.e. Aloe Vera)

    • “Michael Arrington was diagnosed with liver cancer. Doctors       found 17 tumors on his liver. One was the size of a baseball. Doctors       gave the cancer victim 4 to 21 days to live. Six months after Mr.       Arrington began taking only Aloe Vera as a supplement, Doctors examining       the patient’s X-rays could not find [any] trace of the tumors.”
      http://stopcancer.com/deathbed1.htm

Aloe Vera is another one of those natural plants that is very difficult to process correctly to maintain its cancer-fighting properties. The FDA shut down one of the top aloe vera manufacturers because they had a 94% cure rate on terminal cancers.
Aloe Arborescens


High Dose Vitamin C

    • “Basically, the vitamin C is transported to the lungs in the       blood where it is oxidized. It then is transported to the cells where it       diffuses to the mitochondria and delivers its oxidation potential,       powering the respiratory chain, and cycle repeats. It should be noted       that there have been many reports where mega doses of vitamin C have been       attributed to causing the shrinking of cancer tumors.”
      http://www.krysalis.net/cancer.htm       (also used above)

It is always best to take MSM whenever you take Vitamin C or any other protocol that kills microbes. Here is an article on how to use MSM with Vitamin C, regardless of the dose of Vitamin C you take!!
MSM-Vitamin C protocol

http://www.cancertutor.com/Other/ShrinkTumors.html

NaturalNews) The omega-3 essential fatty acid known as docosahexaenoic acid (DHA) is more effective at reducing the size of breast cancer tumors than the chemotherapy drug cisplatin, and can also reduce that drug’s harmful side effects, reports a new study published in the journal Cell Division.

“Our results suggest a new, fruitful drug regimen in the management of solid tumors based on combining cisplatin and possibly other chemotherapeutics with DHA,” said researcher A.M. El-Mowafy of Egypt’s Mansoura University. “DHA elicited prominent chemo-preventative effects on its own, and appreciably augmented those of cisplatin as well. Furthermore, this study is the first to reveal that DHA can obliterate lethal cisplatin-induced nephrotoxicity [kidney damage] and renal tissue injury.”

Researchers injected a group of mice with breast cancer cells, then treated them with either 125 or 250 milligrams per kilogram of DHA, a regular dose of cisplatin, a mix of cisplatin and 125 milligrams per kilogram of DHA, or a placebo. They recorded tumor size and blood levels of white blood cells, C-reactive protein (CRP) and MDA at the start of the study and again after 20 days.

CRP is a marker of inflammation, a known risk factor for tumor growth. MDA is a marker of lipid peroxidation, which signifies high levels of the free radicals that can lead to cancer. Elevated white blood cell counts are also associated with tumor growth.

The researchers found that compared with mice that had never been injected with cancer cells, the injected mice underwent a significant increase in levels of CRP, MDA and white blood cells. Elevated levels of CRP and white blood cells were significantly correlated with increased tumor size.

Levels of white blood cells, CRP and MDA were all lower in animals that had been treated with either DHA, cisplatin, or a combination, however.

In animals who received 125 milligrams per kilogram of DHA, tumor growth was 38 percent less than in animals who received a placebo. Animals receiving cisplatin had 55 percent less tumor growth, while those treated with 250 milligrams per kilogram of DHA had 79 percent less. The combination of DHA and cisplatin not only reduced tumor growth by 81 percent compared with a placebo, it also returned white blood cell counts to normal levels. The 250 milligram per kilogram dose of DHA was nearly as effective at restoring a normal white blood cell count as the DHA-cisplatin combination.

“The chemoprevention elicited by DHA was dose-dependent, and appeared to be mediated by reduction of leukocytosis [elevated white blood cell count], oxidative stress, and replenishing of endogenous antioxidant machinery,” the researchers wrote. “Most strikingly, a strong anti-inflammatory effect was produced.”

In a second experiment, researchers treated rats with cisplatin, which is known to produce potentially lethal kidney damage. Half the rats were also given a 250 milligram per kilogram dose of DHA, while the other half was given nothing.

All the rats that received only cisplatin died from kidney toxicity. Among animals given both the drug and the omega-3, however, only 12 percent developed lethal kidney damage.

DHA is found primarily in fatty cold-water fish, such as salmon, anchovies, herring, mackerel and sardines, but it can also be found in certain vegetable oils. It is believed to play a critical role in the development of the nervous system, particularly the brain and retina.

A number of studies have linked omega-3s such as DHA to a wide variety of health benefits, including reduced risk of cancer and heart disease. A recent study showed that a diet high in omega-3s reduced the risk of prostate cancer even in men with a genetic predisposition for the disease.

Learn more: http://www.naturalnews.com/026784_DHA_cancer_blood.html#ixzz2YTfe1AUb

 

Other healing ways

Tumors tend to grow faster from use of supplement in general, mostly the additive, such as calcium phosphate, calcium, dipotassium phosphate, dicaclium phosphate and the like. Supplements that contain oils, such as vitamin A or E, tends to clog up liver from the liver’s inability to uptake oil. The idea of calcium causing cancer growth and the use of phosphate is well known in alternative cancer treatment for quite some time. This why I ended up making my own supplements or getting them without any additives. In any instances, cancers tend to uptake sodium and they tend to calcify, and hence this is how cancer protects against immune system from eating up the cancer cells.

In cancer treatment, and I don’t follow any popular alternative cancer treatment of today, because it kills people too quickly. There are a couple of ground rules that is most important, avoid fructose, they are the worse of the sugar. It feeds on cancer and they proliferate many fold within hours of taking them. After fructose, it is sucrose, or common table sugar. If cancer really gets bad, then it has to be a ketogenic diet, or a low carbohydrate diet. I have seen a couple of people just died this year believing juicing of fruits will cure them. Apparently they overlooked the fact that it made them diabetic, and the cancer feeds directly on fructose as their primary food.

There are a couple of exceptions on what fruits to eat, the apple is one exception, as their fructose are low, sugar in apple sometimes bind to an anticancer agent, such as quercetin. That’s just about the only fruit that’s safe to eat, including the apple seeds, which contains tiny amounts of laertrile, but not necessarily the apple peels, as they contain flouride. Apples can be eaten as whole, chopped or mashed, but never in apple juices, which tends to cause blood sugar spikes and drink plenty of water.

Obviously the hardening of the cancer is from two things, cancer and sodium. Cancer cells tend to have very high sodium, while normal cells do not. The sodium antagonists is the intracellular fluid mineral called potassium, preferably in the form of potassium citrate. Citrate forms chelate calcium, which removes the calcium protective shell that protects calcium, while reducing the edema as potassium displaces sodium. Obviously, table salt has to be avoided as these lead to edema. There are other potassium I am currently looking at but have trouble obtaining them (budget!) but they are potasium acetate, which may block glycolysis, potassium gluconate, which promotes uptake of sugar to calcium, but using the potassium to kill the cancer by displacing the sodium easier. However, for this condition in particular it’s likely to be the potassium citrate.

The dose for potassium citrate in a cup of warm water is 1/2 teaspoon three times a day after meals. The issue of alkalization only occurs if the potassium citrate is taken only after meals.

As for the iodine tincture mentioned, the iodine tincture I used is the Lugol’s solution, but I am also exploring hydrogen iodide, made from hydrazine, which that in itself is a long story. The only issue that has spooked me for years is the ability for the body to uptake the iodine since iodine excretion is high in itself. The Lugol’s solution I may use 2 drops three for four times a day, preferably three, initially. It is taken in a 1/2 cup of warm water, empty stomach. To help iodine uptake, I believe is linked to the body’s available vitamin D. The one down side of the vitamin D is it’s problem of increasing blood calcium. The only way around this is simply to go out in the sun for 20 minutes, much like sunbathing, twice a day. This should allow the body to produce roughly 10,000 to 20,000 ilu. a day, without the supplements and with no worry of calcium fueling the cancer growth. If there is no sun in the area, perhaps a UV tanning briefly, equivalent to sun exposure of 20 minutes, twice a day. Some aloe vera oil applied thinly may help prevent burns associated with UV exposure. The reason why iodine and vitamin D is so closely linked to cancer is that cancer retention is highest in the month of July, followed by August, while iodine excretion is also minimum at the same time. Hence, the body can utilize the iodine if the body produces sufficient amount of vitamin D. Assuming that it’s not possible to get the sun, or get UV tanning, then perhaps I might consider vitamin D3, but not over 5000 i.u. as the limit and take plenty of potassium citrate, and citric acid to chelate out plenty of calcium. If citric acid can’t be obtain, I might consider the use of lemon juice with plenty of water.

As far as digesting and shrinking of tumor is concerned, they grow simply because the body can’t see the tumor. This can be reversed by allowing them to see, using either of the two things, separately or together. Tannic acid or EGCG (which is actually green tea extract). A green tea extract contains around 50 to 55% EGCG – epigallocatechin gallate). The problems about the use of tannic acid and green tea extract is the constipation, at which I will add sorbitol, which is a laxative, tastes sweet, but also kills cancer cells on contact, in large dose of course, but in this remedy I don’t use this to kill cancer cells. I used it sufficient amount NOT TO CAUSE CONSTIPATION. People with cancer tends to swing two ways. The most common is constipation. The second one which is rarer is diarrhea. Diarrhea from me is relatively easier to treat, which I will use maybe 1/4 teaspoon of humic acid powder, 1/2 teaspoon of fulvic acid powder, or just 1/4 teaspoon of tannic acid, or 1/2 teaspoon of green tea extract powder. In my own experience, tannic acid is the more powerful cancer supplement almost twice more ten that of EGCG, and the dose is confined between 1/4 to 1/2 teaspoon of tannic acid per dose with 1/2 teaspoon of sorbitol usually twice to three times a day in cup of warm water. The dose for EGCG is roughly 1/2 teaspoon on the average to 3/4 teaspoon, with sorbitol again. Should they caused constipation, the potassium citrate might be taken about 1/2 teaspoon four times a day, taken separately at least 30 minutes or more apart. What the tannic acid does is it goes into the bloodstream and make the slippery covering of the cancer, which makes them invisible visible, but deactivating them, basically converting them into leather, or just denaturing the proteins. I read somewhere that if the tannic acid drinks are somewhat acid, the uptake of tannic acid or green tea absorption into the bloodstream is enhanced, but haven’t tested out that theory. To make them acidic, is to add a small squeeze of lemon juice, or some malic acid, or some apple cider vinegar.

In cancer remedy it is most important that digestive enzymes is used. Taken on empty stomach, it goes in the bloodstream so that the digestive enzyme can digest the tumor. The body can actually produce sufficient enzymes to at least digest them by following one simple rule: don’t eat dinner. This makes a person hungry as digestive juices is increased, then it goes into the bloodstream and begin doing its work.

Dietary control is most important. Basically my rule is to avoid most fruits anyway, except for vegetables and carrots. I like carrots because of beta carotene, which is vitamin A and don’t cause liver damage. The other is to avoid completely all vegetable oils including olive oils. The one exception I have never had any complaints in cancer people is the use of coconut oil, which is a monounsaturated fatty acid, and it is this form that doesn’t do liver damage, while krill oil, salmon fish oil, omega 3, polyunsaturated fats tend to do damage to the liver. While omega 3 and other oils such as this maybe good, it is not good for cancer as the liver is already toxic and it can’t handle any more additional oils to cause fatty liver.

The supplements I tend to use is pure ascorbic acid with baking soda, or sodium ascorbate. The reason why baking soda I am not too worry about is it is very alkaline, and when taken together with potassium citrate do not add to more sodium to the cancer cells. What adds more sodium to the cancer cells is the chloride, chlorine, fluoride, which tends to cause sodium retention to increase.

Finally also used lysine as the main element to raise white blood cells by twice that amount, and also eating some pumpkin seeds. Salted pumpkin seeds and fried ones are a problem, just ordinary unsalted pumpkin seeds without and use of vegetable oils tend to raise them. Iodine can also be uptake more easily with kelp or seaweed, but with processing today, I am not sure if they have any iodine left. In Thailand, the popular delicacy is fried seaweed, and tends not to have any iodine, and hence unhealthy.

The lysine supplements I used is taken 1000 mg four to seven times a day for the first couple of days, whereby maybe reduce to four times a day if conditions improve. This will raise white blood cells. I have as yet need to find out what amino acid raises red blood cells, but don’t have sufficient time to do that.

Connie’s comments: Protect your eyes from the sun and not sun bath between 11am to 2pm. Wash your greens/veggies and fruits with salt water or diluted vinegar. Choose organic produce if you can. Avoid sugar and overcooked foods. Controlled fasting can be done once a month for healthy people.

Avoid sugar/soda/processed foods and get good sleep. Avoid anxiety and dwelling on problems, be happy and dance.

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