Less melanin in white people leads to less folate for blood production

Less melanin in white leads to less folate and more melanin in dark colored skin people affects Vitamin D and Calcium absorption

White people must get sunshine to help in folate absorption (folic acid – important nutrient for the blood ) while dark-colored skin people must eat whole foods rich in Vitamin D3 , calcium , omega 3 , Vitamin K2 and magnesium to protect them from diseases related to the heart and circulation/vascular system.

As a result, depression is prevalent among whites while circulatory health issues are common among dark colored skin.

See your doctor for more preventive measures.

Connie Dello Buono

The color of skin is influenced by a number of pigments, including melanin, carotene, and hemoglobin. Recall that melanin is produced by cells called melanocytes, which are found scattered throughout the stratum basale of the epidermis. The melanin is transferred into the keratinocytes via a cellular organelle called a melanosome (Figure 5.7).

This figure consists of two diagrams side by side. The right diagram shows development of light colored skin; the left shows development of dark-colored skin. In both, a brown melanocyte sits at the border between the dermis and epidermis. The melanocyte has a large nucleus and six finger-like extensions. These reach between cells of the stratum basalis. Sections of the extensions detach and travel through the skins. These are melanosomes. In the left diagram, both the melanocyte and melanosomes contain melanin particles, shown as dark dots. Melanosomes travel upwards to outer skin layers, releasing melanin. As a result, keratinocytes in the left diagram contain several melanin particles that darken skin color. In light colored skin, the melanocyte contains no melanin. It still releases melanosomes into upper layers of the skin; however, these melanosomes contain no melanin. Therefore, the skin does not darken and remains light.
Figure 5.7. Skin Pigmentation
The relative coloration of the skin depends of the amount of melanin produced by melanocytes in the stratum basale and taken up by keratinocytes.

Melanin occurs in two primary forms. Eumelanin exists as black and brown, whereas pheomelanin provides a red color. Dark-skinned individuals produce more melanin than those with pale skin. Exposure to the UV rays of the sun or a tanning salon causes melanin to be manufactured and built up in keratinocytes, as sun exposure stimulates keratinocytes to secrete chemicals that stimulate melanocytes.

The accumulation of melanin in keratinocytes results in the darkening of the skin, or a tan. This increased melanin accumulation protects the DNA of epidermal cells from UV ray damage and the breakdown of folic acid, a nutrient necessary for our health and well-being.

In contrast, too much melanin can interfere with the production of vitamin D, an important nutrient involved in calcium absorption.

Thus, the amount of melanin present in our skin is dependent on a balance between available sunlight and folic acid destruction, and protection from UV radiation and vitamin D production.

It requires about 10 days after initial sun exposure for melanin synthesis to peak, which is why pale-skinned individuals tend to suffer sunburns of the epidermis initially. Dark-skinned individuals can also get sunburns, but are more protected than are pale-skinned individuals. Melanosomes are temporary structures that are eventually destroyed by fusion with lysosomes; this fact, along with melanin-filled keratinocytes in the stratum corneum sloughing off, makes tanning impermanent.

Too much sun exposure can eventually lead to wrinkling due to the destruction of the cellular structure of the skin, and in severe cases, can cause sufficient DNA damage to result in skin cancer.

When there is an irregular accumulation of melanocytes in the skin, freckles appear. Moles are larger masses of melanocytes, and although most are benign, they should be monitored for changes that might indicate the presence of cancer (Figure 5.8).

Five photos of moles. The three upper photos show moles that are small, flat, and dark brown. The bottom left photo shows a dark black mole that is raised above the skin. The bottom right photo shows a large, raised, reddish mole with protruding hairs.
Figure 5.8. Moles
Moles range from benign accumulations of melanocytes to melanomas. These structures populate the landscape of our skin. (credit: the National Cancer Institute)
 
 

SKIN DISCOLORATION

The first thing a clinician sees is the skin, and so the examination of the skin should be part of any thorough physical examination. Most skin disorders are relatively benign, but a few, including melanomas, can be fatal if untreated. A couple of the more noticeable disorders, albinism and vitiligo, affect the appearance of the skin and its accessory organs. Although neither is fatal, it would be hard to claim that they are benign, at least to the individuals so afflicted.

Albinism is a genetic disorder that affects (completely or partially) the coloring of skin, hair, and eyes. The defect is primarily due to the inability of melanocytes to produce melanin. Individuals with albinism tend to appear white or very pale due to the lack of melanin in their skin and hair. Recall that melanin helps protect the skin from the harmful effects of UV radiation. Individuals with albinism tend to need more protection from UV radiation, as they are more prone to sunburns and skin cancer. They also tend to be more sensitive to light and have vision problems due to the lack of pigmentation on the retinal wall. Treatment of this disorder usually involves addressing the symptoms, such as limiting UV light exposure to the skin and eyes.

In vitiligo, the melanocytes in certain areas lose their ability to produce melanin, possibly due to an autoimmune reaction. This leads to a loss of color in patches (Figure 5.9). Neither albinism nor vitiligo directly affects the lifespan of an individual.

This photo shows the back of a man’s neck. There is a large, discolored patch of skin at the base of his hairline. The discolored area extends over the ears onto the cheeks, toward the front of the face. The man’s head and facial hair are mostly gray, but white patches of hair are seen above the discolored skin.
Figure 5.9. Vitiligo
Individuals with vitiligo experience depigmentation that results in lighter colored patches of skin. The condition is especially noticeable on darker skin. (credit: Klaus D. Peter)
 

Other changes in the appearance of skin coloration can be indicative of diseases associated with other body systems. Liver disease or liver cancer can cause the accumulation of bile and the yellow pigment bilirubin, leading to the skin appearing yellow or jaundiced (jaune is the French word for “yellow”). Tumors of the pituitary gland can result in the secretion of large amounts of melanocyte-stimulating hormone (MSH), which results in a darkening of the skin. Similarly, Addison’s disease can stimulate the release of excess amounts of adrenocorticotropic hormone (ACTH), which can give the skin a deep bronze color.

A sudden drop in oxygenation can affect skin color, causing the skin to initially turn pale (white), a condition called pallor. With a prolonged reduction in oxygen levels, dark red deoxyhemoglobin becomes dominant in the blood, making the skin appear blue, a condition referred to as cyanosis (kyanos is the Greek word for “blue”).

This happens when the oxygen supply is restricted, as when someone is experiencing difficulty in breathing because of asthma or a heart attack. However, in these cases the effect on skin color has nothing do with the skin’s pigmentation.

INTERACTIVE LINK

This ABC video follows the story of a pair of fraternal African-American twins, one of whom is albino. Watch this video to learn about the challenges these children and their family face. Which ethnicities do you think are exempt from the possibility of albinism?

Calcium and magnesium balance 60:40 ratio

Cleveland Ohio health data: infant mortality and heart disease

cleve-infant-whycleve-infantcleveland-ohio-num-1-heart-diseaseohio-1ohio-2

Why is there high rate of infant mortality among hispanics/latino in Wards 3, 11,14, 15 and 17?

Why is heart diease higher in Cleveland Ohio than the nation?

What quality programs were implemented to reduce heart health risks and infant mortality risks?

ohio-quality

Blue bions – high energy whole foods kills T-bacilli cancer cells

Orgone Experiments

The new observations with a superior microscope also validate the orgone experiments of Wilhelm Reich who described similar microbial processes in the 1940’s (The Cancer Biopathy. Farrar, Straus and Giroux, NY, 1973). He accepted only ‘terminal’ cancer patients and treatment was free. Commonly pain diminished, blood and weight improved, and tumours shrank or disappeared. Despite this, the patients died. From this he concluded that tumours are not an important part of the disease.

Reich’s work was so threatening to the medical establishment that all of Reich’s published books were burned and equipment destroyed under FDA supervision. Because he maintained that a court was not a proper place to discuss a scientific theory, he died 1957 in an US jail (www.wilhelmreichtrust.org/biography.html).

Bion

His most important achievement, which I regard as the greatest scientific discovery of the last century, was the bion as the basic unit of biological life. He found that every kind of food and vegetable matter when heated to incandescence and then dropped into a sterile nutrient solution evolved into round moving or pulsating bions that appeared blue in dark-field or when viewed with a fluorescent microscope. Reich sacrificed fine structure details and observed mainly at 3000 – 5000x in order to better see colour and movements.

Higher the vitality of the entity – blue Bions

The stronger the blue colour, the higher the vitality of the entity. Under these conditions healthy erythrocytes have a blue appearance while dead ones are black. According to Reich this blue glimmer is the characteristic of bio-energy or life-force which he called orgone. It is present not only in all living matter but also in water and air. Orgone originates in the sun and is transmitted by sunlight. Bions, the biological units of orgone, may gradually aggregate and evolve into amoeba-like or protozoan structures.

Degenerating proteins as T-bacilli

Reich described another formation derived from degenerating proteins as T-bacilli, which stands for the German ‘Todes Bacilli’ or ‘death bacilli’. These can easily be cultivated from cancer tissue, the blood of patients with cancer or precancerous conditions, and also from degenerating blood. Injected in high doses they can kill mice within 24 hours, in lower doses they produce cancerous growths. They are black, lancet shaped and of similar size as described by Olbrich.

Blue bions paralysed and killed the black T-bacilli

Also interesting is the observation that the blue bions paralysed and killed the black T-bacilli and even the much bigger proteus bacilli. In the same way strongly blue erythrocytes killed T-cells and pathogenic bacteria, but thereby the erythrocytes lost some of their blue colour, indicating that their vitality diminished in the process.

When strongly charged red blood cells entered a tumour, cancer tissue started to disintegrate into non-motile T-bodies. But in addition also the red blood cells disappeared and only T-bodies remained visible. The tumour developed large cavities which filled up with T-bodies. Macroscopically the originally blood-red cavities turned into a rust brown colour from the disintegrating tumour and blood cells.

Sunlight

Reich observed that weak erythrocytes could be charged to become strongly blue and vital by using sunlight, and especially his orgone accumulator which concentrates orgone from the atmosphere. Some healers have the ability to channel strong bio-energy with their hands and to some degree even remotely with their mind.

Preventable hospitalizations among racial groups, 2003

In 2003, racial and ethnic dis-parities existed in the rates of preventable hospitalizations, with blacks generally having the highest rates and Hispanics the second highest rates.

  • The disparities were greatest for hospitalizations for chronic conditions such as diabetes, hypertension, and asthma. Compared with non-Hispanic whites, rates of admission for these conditions were about 3 to 5 times greater among blacks, and approximately 2 to 3 times greater among Hispanics.
  • Compared with non-Hispanic whites, blacks had higher rates of preventable hospitalizations for 15 of 17 indicators, and Hispanics had higher rates of preventable hospitalizations for 14 of 17 indicators.
  • Asians were less likely than non-Hispanic whites to be ad-mitted for preventable hospitali-zations, with 9 of 17 indicators being lowest in Asians.
  • Blacks had the highest rates of preventable hospitalizations for all indicators related to diabetes and circulatory diseases. Hospi-talization rates for hypertension and for diabetes without compli-cations were 5 times higher for blacks than for non-Hispanic whites. Hospitalization rates for pediatric asthma, adult asthma, perforated appendix, dehydra-tion, and low birth weight were also highest among blacks.
  • Hispanics had the highest rates of admission for elderly asthma, pediatric gastroenteritis, and urinary tract infection.
  • Admissions for asthma among patients 65 and older were 1.8 times more likely for Asians than for non-Hispanic whites—the only indicator where hospitaliza-tion rates were higher in Asians.

This Statistical Brief is based on PQI Version 2.1, revision 3. This PQI version includes measures for hospital admission rates for the following 16 ambulatory care-sensitive conditions:

– Lower-extremity amputations among patients with diabetes (a specific, serious, long-term complication of diabetes)

– Diabetes, long-term complications (i.e., chronic conditions such as renal, visual, neurological, and circulatory disorders, including lower-extremity amputations)

– Diabetes, short-term complications (i.e., acute conditions such as diabetic ketoacidosis, hyperosmolarity, and coma)

– Uncontrolled diabetes without complications

– Angina without procedure

– Hypertension

– Congestive heart failure

– Pediatric asthma

– Adult asthma

– Chronic obstructive pulmonary disease

– Pediatric gastroenteritis

– Perforated appendix

– Urinary tract infections

– Dehydration

– Bacterial pneumonia

– Low-birth weight

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Hormone therapy for prostate cancer may pose a risk for black men

By Lateshia Beachum

Black men treated with hormone therapy for prostate cancer may have a higher risk of death than white men undergoing the same therapy, according to a new study. But the deaths aren’t actually caused by prostate cancer.

Androgen deprivation therapy, or ADT, is a hormone treatment that shrinks prostate tumors. Researchers from the Brigham and Women’s Hospital in Boston found that black men undergoing the therapy had a 77 percent higher risk of death than non-black men.

Researchers at Brigham and Women’s Hospital looked through medical records of about 1,500 men from the Chicago Prostate Cancer Center with low- or intermediate-risk prostate cancer who were treated with ADT. About 7 percent were black.

The small study, in the journal Cancer, found that black men were usually younger, treated later and more likely to have other health issues.

Black men had a higher incidence of death after only four months of hormone therapy, the researchers found, but none of the causes of death in the study were actually prostate cancer.

Konstantin Kovtun, one of the lead researchers for the study, said the deaths seem to be related to cardiovascular health issues that existed prior to a cancer diagnosis. “African American men have an onset of cardiovascular problems that are linked to ADT use,” he said.

The study noted that earlier research has found shorter survival rates among intermediate- to high-risk prostate cancer patients using ADT because of an increased risk in fatal heart attack.

Jonathan Simons, president and chief executive officer of the Prostate Cancer Foundation, said the study was interesting but needs to be followed by more research, including a closer examination of the causes of death. “We don’t know the real reason for the number of deaths,” he said.

Black men typically fare worse with prostate cancer than white men, Simons said, and the reasons for the differences are understudied and poorly understood.

“You can have a man of European and a man of African descent,” he said. “Even if they get the same health care, insurance and doctors, black men still have it worse with prostate cancer.”

Kovtun hopes that the study helps inform decisions about treatment.

“If I was a physician and I had the data now available for the study, I would be more careful in using ADT for African American men,” he said.