Solar in California by 2045

With solar roof panels, you can be rest assured that you will not be experiencing an average yearly increase of power bills from PGE by an estimated 10%. There are currently only about 10% of homes are powered by solar and will soon double by end of this year especially for homes with electric cars, near schools and commercial establishments. California also leads the nation in the number of homes which have solar panels installed, totaling over 230,000. Many were installed because of the Million Solar Roof Initiative.

The U.S. installed 3.8 gigawatts (GW) of solar PV capacity in Q3 2020 to reach 88.9 GW of total installed capacity, enough to power 16.4 million American homes.

The Net Metering Program began to help set up homes for solar. New smart meters were ordered by the state government to be changed for each home in California by the utility companies. The new meters track positive credits and energy usage when energy is being produced from the home.

The first two projects to use net metering were an apartment complex and a solar test house in Massachusetts in 1979. Minnesota is commonly cited as passing the first net metering law, in 1983, and allowed anyone generating less than 40 kWh to either roll over any credit to the next month, or be paid for the excess.

San Diego Gas & Electric switched over to NEM 2.0 on June 29, 2016, and Pacific Gas & Electric on December 15, 2016. Southern California Edison didn’t reach its cap and switched over on July 1, 2017. In short, since July 1, 2017, all major investor-owned power utilities in California operate under NEM 2.0.

There is a no cost solar equipment under a Power Purchased Agreement, PPA, where the customer agreed to a flat rate with annual escalator of 2.9, about half a penny each year.

There is also a solar financing, where you can add the new roof loan and or Tesla battery in the solar financing. With solar financing, you can avail of the 26% tax incentive be subtracted from your solar financing amount if you qualify based on your income.
The investment tax credit (ITC), also known as the federal solar tax credit, allows you to deduct 26 percent of the cost of installing a solar energy system from your federal taxes. The ITC applies to both residential and commercial systems, and there is no cap on its value.

California Sets Goal Of 100 Percent Clean Electric Power By 2045Solar panels are mounted on the roof of the Los Angeles Convention Center on September 5. The state’s governor has signed a landmark bill setting a goal of 100 percent clean energy for the state’s electrical needs, by the year 2045.

Email and text Connie to have solar powered house in California, Texas, New Mexico, Florida, Hawaii, Colorado, Arizona and other states at 408-854-1883 , motherhealth@gmail.com . Your savings can be used to power up your retirement savings, estate creation and asset protection.

Increase your home value with solar, no house lien, transferable agreement, warranty and maintenance from solar carriers, 0 upfront cost, get new roof added to solar financing, and more benefits. Own it after 5 years , financing for 10 or 25 years or just used it with PPA for 25 years.

You may add in the financing, the Tesla battery ($15k cost) or not in California.

Save the planet and save for your retirement in one with solar. We are hiring solar reps and wealth managers with life insurance license.

Connection between light sensitive nerve cells in eyes and brain that regulate mood

Buzzing the vagus nerve just right to fight inflammatory disease and Vit D for chronic headache

Light and anxiety

Underappreciated External Triggers: Fluorescent Lights and Things …

Oct 3, 2008 – This is the Part V of V Share Posts pertaining to categories of “triggers” that can “get the ball rolling” or accelerate your anxiety. If you’re reading this post first, you may want to go back and read Understanding the Vicious Cycle of Panic,” first to appreciate the idea that while anxiety may appear to come “out …

Light Sensitivity and Anxiety – TheraSpecs

Aug 31, 2017 – In addition, light sensitivity and anxiety is tied to specific conditions. … In fact, migraineurs with light sensitivity between attacks (known as ‘interictal’ photophobia) are more likely to develop feelings of depression, anxiety and stress.

Types of Visual Problems and Anxiety – Calm Clinic

If you find it difficult to see clearly, notice flashes or visual snow, or feel like lights become too bright when you are anxious, it doesn’t necessarily mean there is something wrong with your eyes: these can be part of the body’s natural anxiety response (as inconvenient as they may be). In situations where fear is called for, they …

How Anxiety May Interfere With Your Eyes – Calm Clinic

There are many different ways that anxiety can affect your eyes. The most common ways are: Eye pain and discomfort. Mildly blurred vision. Watery eyes. Light sensitivity. Eye floaters (spots that float across your eye). These are all most common during periods of intense anxiety, and the most common time to experience …

Fluorescent Lights and Anxiety – Stop Anxiety Panic Attack

May 21, 2009 – Have you ever experienced feeling an overwhelming wave of anxiety rapidly wash over you once you have stepped inside a pharmacy, at work or a convenience store? If yes, this is probably because pharmacies and convenience stores are lit with harsh, fluorescent lighting.

Light Therapy for Anxiety and Depression In Your Pocket: Human …

anxiety-gone.com/how-light-therapy-for-anxiety-and-depression-will-change-your-life/

Sep 26, 2017 – With the majority of therapy types costing a pretty penny, it can be easy to get discouraged. However, light therapy for anxiety and depression is one of my favourite forms of treatment and it won’t completely break your bank. Here’s what you need to know.

Anxiety and Sensitive to light: Common Related Medical Conditions

WebMD Symptom Checker helps you find the most common medical conditions indicated by the symptoms Anxiety and Sensitive to light and including Generalized anxiety disorder, Migraine headache (adult) and Panic attack.

Why CFL’s Aren’t Such a Bright Idea | Psychology Today

Sep 15, 2014 – In light of rising environmentally-sensitive brain disorders such as autism and ADHD in children, are energy-saving CFL’s costing us dearly? … a higher visual “load” when processing fluorescentlight, which depletes mental resources and makes the individual more likely to be agitated, disruptive,anxious, …

Mitochondrial disease , anti-oxidants, nutrition test and Dementia

Mitochondrial disease , anti-oxidants, nutrition test and Dementia

From Wikipedia, the free encyclopedia

Mitochondrial disease

Mitochondrial diseases are a group of disorders caused by dysfunctional mitochondria, the organelles that generate energy for the cell. Mitochondria are found in every cell of the human body except red blood cells, and convert the energy of food molecules into the ATP that powers most cell functions.

Mitochondrial diseases are sometimes (about 15% of the time)[1] caused by mutations in the mitochondrial DNA that affect mitochondrial function. Other mitochondrial diseases are caused by mutations in genes of the nuclear DNA, whose gene products are imported into the mitochondria (mitochondrial proteins) as well as acquired mitochondrial conditions. Mitochondrial diseases take on unique characteristics both because of the way the diseases are often inherited and because mitochondria are so critical to cell function. The subclass of these diseases that have neuromuscular disease symptoms are often called a mitochondrial myopathy.

Signs and symptoms

Symptoms include poor growth, loss of muscle coordination, muscle weakness, visual problems, hearing problems, learning disabilities, heart disease, liver disease, kidney disease, gastrointestinal disorders, respiratory disorders, neurological problems, autonomic dysfunction and dementia. Acquired conditions in which mitochondrial dysfunction has been involved are: diabetes, Huntington’s disease, cancer, Alzheimer’s disease, Parkinson’s disease, bipolar disorder,[2][3][4] schizophrenia, aging and senescence, anxiety disorders, cardiovascular disease, sarcopenia, chronic fatigue syndrome.[5]
The body, and each mutation, is modulated by other genome variants; the mutation that in one individual may cause liver disease might in another person cause a brain disorder. The severity of the specific defect may also be great or small. Some minor defects cause only “exercise intolerance”, with no serious illness or disability. Defects often affect the operation of the mitochondria and multiple tissues more severely, leading to multi-system diseases.[citation needed]
As a rule, mitochondrial diseases are worse when the defective mitochondria are present in the muscles, cerebrum, or nerves,[6] because these cells use more energy than most other cells in the body.
Although mitochondrial diseases vary greatly in presentation from person to person, several major clinical categories of these conditions have been defined, based on the most common phenotypic features, symptoms, and signs associated with the particular mutations that tend to cause them.
An outstanding question and area of research is whether ATP depletion or reactive oxygen species are in fact responsible for the observed phenotypic consequences.
Cerebellar atrophy or hypoplasia has sometimes been reported to be associated.

Causes

Mitochondrial disorders may be caused by mutations (acquired or inherited), in mitochondrial DNA (mtDNA), or in nuclear genes that code for mitochondrial components. They may also be the result of acquired mitochondrial dysfunction due to adverse effects of drugs, infections, or other environmental causes (see MeSH).
Nuclear DNA has two copies per cell (except for sperm and egg cells), one copy being inherited from the father and the other from the mother. Mitochondrial DNA, however, is strictly inherited from the mother and each mitochondrial organelle typically contains between 2 and 10 mtDNA copies. During cell division the mitochondria segregate randomly between the two new cells. Those mitochondria make more copies, normally reaching 500 mitochondria per cell. As mtDNA is copied when mitochondria proliferate, they can accumulate random mutations, a phenomenon called heteroplasmy. If only a few of the mtDNA copies inherited from the mother are defective, mitochondrial division may cause most of the defective copies to end up in just one of the new mitochondria (for more detailed inheritance patterns, see human mitochondrial genetics). Mitochondrial disease may become clinically apparent once the number of affected mitochondria reaches a certain level; this phenomenon is called “threshold expression”.
Mitochondrial DNA mutations occur frequently, due to the lack of the error checking capability that nuclear DNA has (see Mutation rate). This means that mitochondrial DNA disorders may occur spontaneously and relatively often. Defects in enzymes that control mitochondrial DNA replication (all of which are encoded for by genes in the nuclear DNA) may also cause mitochondrial DNA mutations.
Most mitochondrial function and biogenesis is controlled by nuclear DNA. Human mitochondrial DNA encodes 13 proteins of the respiratory chain, while most of the estimated 1,500 proteins and components targeted to mitochondria are nuclear-encoded. Defects in nuclear-encoded mitochondrial genes are associated with hundreds of clinical disease phenotypes including anemia, dementia, hypertension, lymphoma, retinopathy, seizures, and neurodevelopmental disorders.[8]

A study by Yale University researchers (published in the February 12, 2004 issue of the New England Journal of Medicine) explored the role of mitochondria in insulin resistance among the offspring of patients with type 2 diabetes.[9] Other studies have shown that the mechanism may involve the interruption of the mitochondrial signaling process in body cells (intramyocellular lipids). A study conducted at the Pennington Biomedical Research Center in Baton Rouge, Louisiana[10] showed that this, in turn, partially disables the genes that produce mitochondria.

Examples of mitochondrial diseases include:

  • Mitochondrial myopathy
  • Diabetes mellitus and deafness (DAD)
    this combination at an early age can be due to mitochondrial disease
    Diabetes mellitus and deafness can be found together for other reasons
  • Leber’s hereditary optic neuropathy (LHON)
    visual loss beginning in young adulthood
    eye disorder characterized by progressive loss of central vision due to degeneration of the optic nerves and retina
    affects 1 in 50,000 people in Finland
  • Leigh syndrome, subacute sclerosing encephalopathy
    after normal development the disease usually begins late in the first year of life, although onset may occur in adulthood
    a rapid decline in function occurs and is marked by seizures, altered states of consciousness, dementia, ventilatory failure
  • Neuropathy, ataxia, retinitis pigmentosa, and ptosis (NARP)
    progressive symptoms as described in the acronym
    dementia
  • Myoneurogenic gastrointestinal encephalopathy (MNGIE)
    gastrointestinal pseudo-obstruction
    neuropathy
    Myoclonic Epilepsy with Ragged Red Fibers (MERRF)
    progressive myoclonic epilepsy
    “Ragged Red Fibers” are clumps of diseased mitochondria that accumulate in the subsarcolemmal region of the muscle fiber and appear when muscle is stained with modified Gömöri trichrome stain
  • short stature
  • hearing loss
  • lactic acidosis
  • exercise intolerance
  • Mitochondrial myopathy, encephalomyopathy, lactic acidosis, stroke-like symptoms (MELAS)
  • mtDNA depletion
  • mitochondrial neurogastrointestinal encephalomyopathy (MNGIE)
    Conditions such as Friedreich’s ataxia can affect the mitochondria but are not associated with mitochondrial proteins.

Mechanisms

The effective overall energy unit for the available body energy is referred to as the daily glycogen generation capacity,[11][12][13] and is used to compare the mitochondrial output of healthy individuals to that of afflicted or chronically glycogen-depleted individuals. This value is slow to change in a given individual, as it takes between 18 and 24 months to complete a full cycle.

The glycogen generation capacity is entirely dependent on, and determined by, the operating levels of the mitochondria in all of the cells of the human body;[14] however, the relation between the energy generated by the mitochondria and the glycogen capacity is very loose and is mediated by many biochemical pathways.[11] The energy output of full healthy mitochondrial function can be predicted exactly by a complicated theoretical argument, but this argument is not straightforward, as most energy is consumed by the brain and is not easily measurable.

Diagnosis

Mitochondrial diseases are usually detected by analysing muscle samples, where the presence of these organelles is higher.

Connie’s comments:  NIH created nutrition tester which test for anti-oxidant levels can be used as my initial experiences with various test results where Dr Oz got 85k high score and I have 41k lower from 2 years ago of 55k.

Email motherhealth@gmail.com to be a nutrition test site with this Raman Spectroscopy tester to suggest nutrition protocols ( AGELOC family which resets gene expression to younger you)  at:

http://www.clubalthea.pxproducts.com

Treatments

Although research is ongoing, treatment options are currently limited; vitamins are frequently prescribed, though the evidence for their effectiveness is limited.[15] Pyruvate has been proposed in 2007 as a treatment option.[16] N-acetyl cysteine reverses many models of mitochondrial dysfunction.[17]. In the case of mood disorders, specifically bipolar disorder, it is hypothesized that N-acetyl-cysteine (NAC), acetyl-L-carnitine (ALCAR), S-adenosylmethionine (SAMe), coenzyme Q10 (CoQ10), alpha-lipoic acid (ALA), creatine monohydrate (CM), and melatonin could be potential treatment options.

Anti-oxidants and colored fruits and veggies are your first defense.

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